Viewing negative mentions of positive regulation of IL2 (H. sapiens) in B cells

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Umetsu et al. (1988)interleukin 2B cellsYet in contrast to allogeneic monocytes, allogeneic B cells failed to induce a rise in the intracellular calcium ion concentration and failed to cause interleukin 2 (IL2) receptor expression in Clone A1.
Prasad et al. (1997)IL-2B cellThe most potent Ag presentation for IL-2 secretion was found to segregate with low-density, B cell-enriched fractions while adherent cells, or purified T cells were unable to support IL-2 production.
Panayotides et al. (1986)IL-2B-cellSome human B-cell malignancies have been reported to react with anti-IL-2 receptor antibodies, but no response to IL-2 has been documented in these cases.
Brown et al. (1983)IL-2B-cellSeveral established primate B-cell lines which do not require IL-2 for growth were able to proliferate in the serum-free medium.
Sauerwein et al. (1985)IL 2B cellsIn the absence of T cells, IL 2 could not activate Ig synthesis, suggesting an indirect effect of IL 2 in the induction of the helper signals for B cells.
Choi and Xia (1987)IL-2B cellsHence, the conventional method using preactivated B cells does not measure BCGF but mostly IL-2.
Bartlett et al. (1989)IL-2B cellIn contrast to the absolute requirement of IL-2 for Th-3.1-induced B cell proliferation, IL-2 was not required for the formation of stable Th-3.1-B cell conjugates or Th-3.1-induced B cell entry into the G1 phase of the cell cycle.
Doepper et al. (2000)IL-2B cellSurprisingly, enhancement of T cell infection by B cell-HIV complexes was observed even in the absence of mitogenic stimuli such as PMA and was independent of the addition of exogenous IL-2.
Mitsui et al. (1991)interleukin 2B cellHigh-affinity interleukin 2 receptors on B cell chronic lymphocytic leukemia cells are induced by phorbol myristate acetate but not by calcium ionophore.
Kasprzak et al. (2010)IL-2B-cellUsing hybridocytochemistry, presence of mRNA for IL-2 could be detected, but not mRNA for IL-22R(alpha) in children with B-cell NHLs.
Alvarez-Mon et al. (1989)IL-2B cellsIn agreement, upon stimulation, B cells expressed Bac-1 antigen but failed to acquire the IL-2 receptor.
Jeong et al. (1986)IL-2B-cellDex did not induce IL-2 production and inhibited IL-2 production induced by Con A, in contrast to the promoting effects of Dex on BIF production, providing further evidence for the independence of BIF and IL-2 production and B-cell stimulation.
Kondadasula et al. (2008)IL-2B cellsAn analysis of PBMCs stimulated with IL-2 indicated that IL-2 exposure induced p-ERK in CD56bright NK cells and CD14+ monocytes, but not in CD3+ T cells or CD21+ B cells.
Chorba et al. (1985)interleukin 2B cellsCM cell cultures and the transformed CBL do not require exogenous interleukin 2, have T cell cytochemical features and mature T helper phenotypes, and exhibit minimal T helper and profound T suppressor activity on pokeweed mitogen-stimulated differentiation of normal B cells.
Darko et al. (1988)interleukin-2B lymphocytesPercent of total lymphocytes labeled as all T lymphocytes, all B lymphocytes, and natural killer cells did not differ in the two groups, nor did mitogen-induced interleukin-2 production.
Kimata and Fujimoto (1994)IL-2B cellsFurther, anti-CD40 mAb plus various cytokines, including interleukin (IL)-1 beta, IL-2, IL-3, IL-4, IL-5, IL-6, IL-10, transforming growth factor-beta, low molecular weight B cell growth factor, and interferon-gamma, did not induce IgA1 and IgA2 production by sIgA1- and sIgA2- B cells, respectively.
Inoue et al. (1993)IL-2B cellsSome patients showed no increase in expression of IL-2 receptors on B cells with SAC stimulation, which may be due to impaired maturation stages.
de la Calle-Martín et al. (1992)IL-2B lymphocytesIn the presence of Mo, PWM stimulation of T lymphocytes (highly depleted of B lymphocytes) induces as much IL-2 mRNA as phytohemagglutinin (PHA), but results in higher and persistent IL-2 levels in culture supernatants despite the concomitant T cell mitogenesis, suggesting that PWM-activated T cells do not utilize the IL-2 they produce.
Goedert et al. (2000)hrIL-2B cellsWe could demonstrate that canine B cells react on hrIL-2 alone by proliferation and IgG production but not by IgE secretion, whereas activation with hrIL-4 induced proliferation and mainly IgE production.