Viewing affirmative mentions of gene expression of IL2 (H. sapiens) in memory T cells

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Strauss et al. (1998)interleukin-2memory T cellsK+ currents of encephalitogenic memory T cells decrease with encephalitogenicity while interleukin-2 (IL-2) receptor expression remains stable during IL-2 dependent cell expansion.
Mallard et al. (2004)IL-2memory T cellsMoreover, the majority of central memory T cells (CD45RAlowCCR7dull) secreting cytokines in response to an EBV epitope produces both IL-2 and IFN-gamma, whereas effector memory CD8 cells (CD45RAdullCCR7-) found in EBV, CMV, or Melan-A memory pools are mostly composed of cells secreting exclusively IFN-gamma.
Krummel et al. (2010)IL-2memory T cellsis predominantly produced by effector memory T cells, IL-2 is predominantly produced by central memory T cells.
Kruse et al. (1993)lymphokinememory T cellsThese observations suggest that the decrease in the proportion of some T-cell subsets and the absence of CD45RO memory T cells may be responsible for the diminished lymphokine production of cord blood lymphocytes.
Do et al. (2006)IL-2memory T cellsIn vitro-generated T cell lines and freshly isolated CD4+CD45R0+ primed/memory T cells from 77C-->G individuals aberrantly expressed CD45RA isoforms and showed enhanced proliferation and IL-2 production when stimulated with anti-TCR/CD3 mAb or Ag.
Schulze-Koops et al. (1998)IL-2memory T cellsFreshly isolated memory T cells primarily produced IL-2 and small amounts of IL-4 and IFN-gamma after stimulation in vitro.
Guerreiro et al. (2010)IL-2memory T cellsBased on their cytokine profiles, six different EBV-specific T-cell subsets could be distinguished with TNF-single or TNF/IL-2-double producing cells expressing the highest CCR7 levels resembling early-differentiated memory T cells.
Roestenberg et al. (2009)interleukin-2memory T cellsIn this model, we identified the induction of parasite-specific pluripotent effector memory T cells producing interferon-gamma, tumor necrosis factor alpha, and interleukin-2 as a promising immunologic marker of protection.
Liopeta et al. (2009)rhIL-2memory T lymphocytesIn this study using normal peripheral T lymphocytes stimulated either through the TCR-CD3 complex or the TCR-CD3 and the CD28 molecule, we show that IL-10 is produced mainly by memory T lymphocytes after either way of stimulation and is drastically inhibited (70-90%) by cAMP elevating agents. cAMP mediated inhibition was reversed by the use of the specific PKA inhibitor Rp-8-Br-cAMP but not by the addition of exogenous rhIL-2, indicating that the inhibitory effect depends on PKA activation and is not secondary to IL-2 inhibition.
Kondo and Takiguchi (2009)IL-2memory T cellsCCR4(+)CD8(+) T cells effectively produced IL-4, IFN-gamma, IL-2 and tumor necrosis factor-alpha, indicating that they are memory T cells having the ability to secrete type 1 and type 2 cytokines.
Oswald-Richter et al. (2004)IL-2memory T-cellsTo analyze their capacity to proliferate and secrete IL-2 upon TCR triggering, Treg and memory T-cells were sorted into highly purified populations by flow cytometry.
Davis et al. (2001)IL-2memory T cellsRheumatoid synovial CD4+ T cells exhibit a reduced capacity to differentiate into IL-4-producing T-helper-2 effector cells CD4+ memory T cells (Tm) from rheumatoid arthritis peripheral blood (RAPB) or peripheral blood from normal donors produced IL-2, whereas fewer cells secreted IFN-?
Dooms and Abbas (2006)IL-2memory T-cellHere, we review evidence and show novel data on the role of the cytokines interleukin-2 (IL-2) and IL-7 and the costimulator CD28 in CD4+ memory T-cell development.
Santamaria Babi et al. (1995)lymphokinememory T cellTaken together, these observations demonstrate the correlation of CLA expression on circulating memory T cells and disease-associated memory T cell responses in cutaneous hypersensitivity, and they suggest the existence of mechanisms capable of sorting particular T cell Ag specificities and lymphokine patterns into homing receptor-defined memory subsets.
Rivino et al. (2010)IL-2memory T cellHowever, IL-2 also has some immunostimulatory effects, which might be induced at higher levels of IL-2 production (Scheffold et al., 2005), because it can promote memory T cell generation (Dooms et al., 2007) and autoimmunity (Waithman et al., 2008).
Walter et al. (1994)IL-2memory T cellsIn contrast to the production of IL-2, IFN-gamma, GM-CSF, and TNF-alpha, the production of IL-4 after co-stimulation with B7-CD28 interaction was restricted to CD45RO+ memory T cells.
Wasik et al. (2000)IL-2memory T cellsThe levels of IL-2 expression in the envelope (env) peptide-stimulated peripheral blood mononuclear cells were increased in children with a slowly progressive disease, concomitant with higher numbers of CD45RO(+) memory T cells and increased proportions of Th1 clones.
Nesbeth and Conejo-Garcia (2010)IL-2TCMThese TCM cells can undergo robust expansion in response to secondary exposure to antigen and secrete copious levels of IL-2, in stark contrast to TEM cells.
Eyrich et al. (2004)IL-2memory T cellsIL-2 was predominantly produced by CD4(+)CD45RA(+) na´ve, whereas IFN-gamma originated mainly from CD8(+)CD45RO(+) memory T cells.
Knoop et al. (2000)IL-2memory T cellsIn contrast, the response of memory T cells to TT was severely blunted both in terms of IL-2 and interferon-y production.
Almanzar et al. (2005)IL-2memory T-cellThe size of the CD8+ memory T-cell population, which grows well and produces interleukin-2 (IL-2) and IL-4, also increases with aging, but this increase is missing in CMV carriers.
Ramduth et al. (2009)IL-2memory T cellsCentral memory T cells produce mainly IL-2 while effector memory cells produce both IFN-?
Kinne et al. (2007)IL-2memory T cellsIL-15, a cytokine of the IL-2 family with chemoattractant properties for memory T cells, is produced by lining layer cells (including M?)
Davis et al. (2001)IL-2memory T cellsCD4+ memory T cells (Tm) from rheumatoid arthritis peripheral blood (RAPB) or peripheral blood from normal donors produced IL-2, whereas fewer cells secreted IFN-gamma or IL-4 after a brief stimulation.
Das and Levine (2008)IL-2memory T cellsTGF-beta inhibits IL-2 production and promotes cell cycle arrest in TCR-activated effector/memory T cells in the presence of sustained TCR signal transduction.
Herndler-Brandstetter et al. (2005)IL-2memory T cellWe have recently described an IL-2/IL-4-producing CD8+CD25+ non-regulatory memory T cell population that occurs in a subgroup of healthy elderly persons who characteristically still have a good humoral response after vaccination.
Kato et al. (1997)IL-2memory T cellsThese results indicate that IL-12 provides the costimulation for IL-2R alpha expression and IL-2-dependent proliferation of memory T cells and Th1 clones stimulated with TCR ligation, whereas naive T cells require B7-2 costimulation for their response to IL-12.
Nanki and E Lipsky (2000)IL-2memory T cells, CD154, and TNF-related activation-induced cytokine (TRANCE) were expressed by 2-20% of rheumatoid arthritis (RA) synovial tissue CD4+ memory T cells, whereas CD4+ cells that produced IL-2, IL-4, or IL-6 were not detected.
Bachmann et al. (2005)IL-2memory T cellsIn contrast, CD62L+ CD127+ central memory T cells most efficiently produced IL-2 and proliferated extensively in vitro and in vivo upon antigenic restimulation.
Nanki and Lipsky (2000)IL-2memory T cellsIL-10, IL-13, IFN-gamma, tumor necrosis factor (TNF)-alpha, LT-alpha, CD154, and TNF-related activation-induced cytokine (TRANCE) were expressed by 2-20% of rheumatoid arthritis (RA) synovial tissue CD4(+) memory T cells, whereas CD4(+) cells that produced IL-2, IL-4, or IL-6 were not detected.
Cush et al. (2007)IL-2memory T cellDuring the primary immune response, we show generation of CD8+ memory T cell precursors expressing lymphoid homing molecules (CCR7, L-selectin) and homeostatic cytokine receptors (IL-7alpha, IL-2/IL-15beta).
Plebanski et al. (1997)lymphokinememory T cellIn altered peptide ligand (APL) antagonism, the concurrent presentation of particular closely related epitope variants can prevent memory T cell effector functions such as cytotoxicity, lymphokine production and proliferation.
Beadling and Slifka (2005)IL-2memory T cellsWhen measured in real time, peptide antigen and the cytokines, interleukin 12 (IL-12) and IL-18, independently regulate the on/off kinetics of protective (interferon gamma, tumor necrosis factor alpha) and immunomodulatory (IL-2, CD40L) cytokine production by activated T cells and memory T cells.
Peng et al. (2006)interleukin-2memory T cellsNef-specific CD4 and CD8 memory T cells that produced intracellular IFN-gamma, interleukin-2, and tumor necrosis factor (TNF)-alpha were assessed by flow cytometry.
Yoh et al. (2000)IL-2memory T cellsIn this study we examine cytokine levels, T-lymphocyte subsets, natural killer NK cells, memory T cells, and the expression of IL-2 receptors in order to better understand the role of bacterial superantigens and cytokines in the pathogenesis of MRSA-associated GN.