Viewing negative mentions of gene expression of FAS (H. sapiens) in T cells

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Document Target Regulator Anatomy Sentence
Lechner et al. (1996)CD95T cellsResting T cells did not express CD95.
Simon et al. (1996)FasT cellsCD4-CD8- T cells from both patients, although highly activated, did not express functional Fas receptors.
Klas et al. (1993)APO-1T cellsAPO-1 is not expressed on cord blood and the majority of resting T cells, but on activated T cells.
Frucht et al. (2001)FasT cellsActivated T cells in these patients failed to express Fas ligand (FasL) in response to IL-2, which may explain the accumulation of T cells with an activated phenotype and a skewed T cell receptor (TcR) Vbeta family distribution.
Soulimani et al. (1999)FasT cellIndeed, stimulation of both populations by ionomycin and phorbol 12-myristate 13-acetate or through the T cell receptor (anti-CD3) leads to a selective lysis of targets transfected with Fas but not of the parental cell line.
Miyawaki et al. (1992)FasT cellsFor CD4+ and CD8+ T cells, Fas Ag was expressed preferentially on CD45RO+ (memory or previously activated) populations, but not on CD45RO- naive ones.
Miyawaki et al. (1992)FasT cellExpectably, neonatal T cell subpopulations, most of which had the naive (CD45RO-) phenotype, expressed little Fas Ag.
Wei and Cowie (2007)FasT cellsFunctional failure of the Fas (located on chromosome 10q24.1) is associated with ALPS Ia, an autosomal dominant inherited disorder usually manifesting in infancy with lymphadenopathy, splenomegaly, autoimmune cytopenias and persistence of CD4/8- ‘double negative’ T cells (3).
Esser et al. (2001)FasT lymphocytesNoninfectious CXCR4-tropic HIV-1 virions, but not microvesicles, partially activated freshly isolated CD4(+) and CD8(+) peripheral blood mononuclear cell T lymphocytes to express FasL and Fas, but not CD69 or CD25 (interleukin-2 receptor alpha) and eventually die via apoptosis starting 4 to 6 days postexposure.
Spinozzi et al. (1998)FasT lymphocytesWe have previously shown that pulmonary T lymphocytes from untreated asthmatic subjects do not express surface Fas receptors nor do they contain Fas mRNA, yet they display normal levels of Fas ligand.
Walker et al. (2006)Fas-LT-cellIn particular, we found that T cells expressing Fas ligand (Fas-L, CD95L) were eight times more vulnerable to apoptosis than those not expressing Fas-L, which suggests that the T-cell apoptosis is induced by overactivation of the T-cell receptor, possibly in the absence of appropriate costimulation.
Spinozzi (1998)Fas receptorT cellsOnce initiated, the mucosal allergic reaction cannot be turned off in atopic individuals because CD4+ allergen-specific T cells lack surface Fas receptor.
Chott et al. (1997)FasIELsImmunohistochemical analyses of granzyme B, perforin, Fas ligand, and tumor necrosis factor-alpha demonstrated these proteins were not expressed by small intestinal IELs in situ.
Brennan et al. (1988)surfaceT-cellWhereas the majority of T cells use alpha and beta chains to form their T-cell receptor, a small minority of T cells, which do not express the CD4 or CD8 surface markers, use other chains termed gamma and delta to form their receptor.