Viewing negative mentions of binding of KLRD1 (H. sapiens) in T cells

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Document Target Regulator Anatomy Sentence
Valge et al. (1988)cell receptorT cellsProtein kinase C is required for responses to T cell receptor ligands but not to interleukin-2 in T cells.
Adams et al. (1987)cell receptorT cellThus, although suppression was specific at the level of expression, the induction phase was non-specific and non-major histocompatibility complex (MHC) restricted and did not involve interaction of antigen with the T cell receptor.
Powell and Horton (2005)cell receptorT cellWhether or not T cell receptor engagement leads to full activation or tolerance is determined by the context in which the antigen is encountered.
Wang et al. (2002)cell receptorT cellAlthough mutations at Site 1 had little affect, several at Site 2 and at the dimer interface hampered the Ly49A.H-2D(d) interaction, with no effect on gross structure or T cell receptor interaction.
Luz et al. (2002)cell receptorT cellThe Asp77Ser mutation that imbues H-2K(bm3)-dEV8 with its alloreactive properties is located beneath the peptide and does not directly contact the T cell receptor (TCR).
Shindo et al. (2008)cell receptorT cellsSimultaneous, but not sequential, ligation of the T cell receptor and OX40 induces CD4(+)OX40(+) T cells to produce far more IL-2, which causes them to proliferate abundantly and differentiate readily into Th1- or Th2-biased effector memory T cells, especially in Allo HSCT recipients.
Benihoud et al. (1997)cell receptorT cellsThis subset, which represent in vivo activated T cells, can spontaneously lyse Fas+ targets by a mechanism that does not need the interaction of the T cell receptor (TCR) with major histocompatibility complex molecule plus antigen.