Viewing negative mentions of binding of CEL (H. sapiens) in T cells

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Document Target Regulator Anatomy Sentence
Zhao et al. (2006)cellT cellBut there has been no direct evidence showing the engagement of gammadelta T cell receptors (TCR) of the induced cells with MICA.
Cobb and Kasper (2005)cellT-cellAdaptive immune responses have long been considered the "territory" of antigenic proteins, whereas carbohydrates are characterized as T-cell-independent antigens that are not typically recognized by the complete adaptive machinery.
Hillert et al. (1991)cellT cellNo association with germline T cell receptor beta-chain gene alleles or haplotypes in Swedish patients with multiple sclerosis.
Martelli et al. (2001)cellT cellsOf note, while T cell receptor-CD3 engagement did not induce either p62(dok) phosphorylation or Crk-L association in Jurkat T cells, it did inhibit CD2-dependent p62(dok)-Crk-L complexes; this TcR-CD3-mediated regulation was dependent upon ZAP70 kinase activity.
Raman et al. (1998)cellT cellIn contrast, CK2 is not activated following cross-linking of the B cell receptor complex or the T cell receptor complex.
Mizuki et al. (1998)cellT-cellNo rearrangement of T-cell-receptor genes was detected.
Roep (1996)cellT-cellHowever, T-cell autoreactivity to islet antigens is not specific for IDDM, and the T-cell target antigens are not specific for beta-cells.
Klenerman et al. (1994)cellT-cellThis was attributed to either failure of the variant epitope to bind major histocompatibility complex class I or failure of T-cell receptors to bind the presented peptide.
Blue et al. (1988)cellT cellThese results indicate that CD4 perturbation can result in two distinct regulatory activities; one involves the regulation of CD3-T cell receptor function, whereas the other is not directly associated with CD3-T cell antigen receptor function.
Hall et al. (1988)cellT-cellNo case showed immunoreactivity with anti-T-cell antibodies.
Weetman et al. (1988)cellT-cellThese results fail to confirm the reported associations between ophthalmopathy and HLA-DR3 and between Graves' disease and the T-cell receptor C beta polymorphism; they also argue against a strong influence of Gm allotypes in Graves' disease since these genes are in linkage disequilibrium with the S alpha polymorphisms.
Indiveri et al. (1985)cellT-cellT and non-T cells were isolated by rosetting with 2-aminoethylisothiuronium bromide-treated sheep red blood cells (AET-SRBC), by nylon-wool filtration, and by positive or negative selection with anti-class II HLA antigens and anti-T-cell monoclonal antibodies.
Matera et al. (1984)cellT cellHowever, the cells did not form E rosettes, nor did they react with the monoclonal antibodies OKT3, OKT4, OKT8 and OKT11 to human T cell surface antigens.
Poppema et al. (1982)cellT-cellIn contrast, the Reed-sternberg cells did not react with any of these anti-T-cell antibodies or with anti-IgM antiserum, but displayed strong membrane and cytoplasmic staining with anti-la antibody.
Bakács and Mehrishi (2010)cellT cellThe model states that foreign peptides inhibit the complementary interactions between the major histocompatibility complexes and T cell receptors.
Stöckl et al. (1996)cellT cellThese observations indicated that CD43 cross-linkage mimics and monovalent mAb 6F5 inhibits interaction of T cell CD43 with a stimulatory ligand on opposing cells, presumably MHC-I.