Viewing negative mentions of gene expression of Il17a (M. musculus) in T cells

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Sonnenberg et al. (2010)IL-17AT cellsIn contrast, subsets of gut-resident NK cells and skin-resident CD4+ T cells are reported to express IL-22 but do not coexpress IL-17A (Satoh-Takayama et al., 2008; Cella et al., 2009; Duhen et al., 2009; Trifari et al., 2009).
Miyamoto et al. (2003)IL-17 proteinT lymphocytesIntracellular IL-17 protein is detected in T lymphocytes from spleen but not in airway macrophages after coculture and stimulation of these two cell types.
Wakita et al. (2010)IL-17T cellsAnalysis of TCR repertoires in TIL-gammadelta T cells showed that circulating gammadelta T cells, but not skin resident Vgamma5(+)gammadelta T cells, produced IL-17.
Markel et al. (2010)IL-17AT cellsCD8+ T cells failed to produce IL-17A even at the high infection dose (Figure 5, B1).
Sutton et al. (2009)IL-17T cellsHere, we show that gammadelta T cells express IL-23R and the transcription factor RORgammat and produce IL-17, IL-21, and IL-22 in response to IL-1beta and IL-23, without T cell receptor engagement.
Lochner et al. (2008)IL-17T cellst+ T cells that did not express IL-17 after 2–3 d.
Church et al. (2010)IL-17-producingT cellsProduction of IL-6 by IL-17-producing CD4 T cells was also examined however most IL-17-producing CD4 T cells failed to produce IL-6 (data not shown).
Church et al. (2010)IL-17-producingT cellsConsidering the importance of IL-23 to the development and maintenance of Th17 cells, it is striking that over 90% and 85% of IL-17-producing CD4 T cells in the SF and blood respectively did not express IL-23R.
Liu et al. (2005)IL-17T cellsIL-17 is made primarily, if not exclusively by T cells, but relatively little is known about how its expression is regulated.
Hamel et al. (2008)IL-17T cellsFurthermore, there was a significant reduction in the PG-specific CD4(+) T cell recall response as well as significantly fewer PG-specific CD4(+) T cells producing IFN-gamma and IL-17, but not IL-4.
Damsker et al. (2010)IL-17T cellsNevertheless, mice lacking IL-17 are not protected from disease and display elevated numbers of IFN-gamma-producing CD4+ T cells, and, in some cases, lack of IFN-gamma does confer resistance.
Lindell et al. (2008)IL-17T cellsAs expected, CD4+ T cells alone (>99.5% pure) produced no detectable IL-4, IL-5, IL-13, IL-17 or IFN?
Marta et al. (2008)IL-17T cellPurified splenic myeloid DC (mDC) from MyD88(-/-) mice expressed much less IL-6 and IL-23, and serum and T cell IL-17 were absent.
Domingues et al. (2010)IL-17T cell+ IL-17+ double positive CD4+ T cell population in the CNS but not in the periphery of Th17 recipients (Figure 4A).
Zou et al. (2010)IL-17-producingT cells-producing CD8+ T cells (Tc1), but not the IL-17-producing CD8+ T cells (Tc17).
Oda et al. (2003)IL-17T cellsPorphyromonas gingivalis antigen preferentially stimulates T cells to express IL-17 but not receptor activator of NF-kappaB ligand in vitro.
Bouguermouh et al. (2009)IL-17T cellsIn this study, we provide strong evidence for an inhibitory role of CD28 in the development of naďve CD4+ T cells into IL-17-producing T helper cells with no modulation of IL-17 expression in memory CD4+ T cells.
Schountz et al. (2007)IL-17T cellsThe T cells from acutely infected deer mice synthesized a broad spectrum of cytokines, including IFN-gamma, IL-4, IL-5, and TGF-beta(1), but not TNF, lymphotoxin, or IL-17.
Suto et al. (2008)IL-17AT cellsThese results suggest that although IL-21–producing CD4+ T cells develop preferentially under Th17-polarizing conditions, the majority of IL-21–producing CD4+ T cells do not produce IL-17A or -17F.
Suto et al. (2008)IL-17AT cellsBy the newly developed intracellular staining of IL-21, we found that although IL-21–producing CD4+ T cells developed preferentially under Th17-polarizing conditions, a considerable number of IL-21–producing CD4+ T cells were negative for intracellular IL-17A and -17F (Fig. 2).
Lee et al. (2008)IL-17T cellsA distinct subset of natural killer T cells produces IL-17, contributing to airway infiltration of neutrophils but not to airway hyperreactivity.
Suto et al. (2008)IL-17AT cellsUnexpectedly, we found that under Th17-polarizing conditions, the majority of IL-21–producing CD4+ T cells did not produce IL-17A and -17F.
Berg (2009)interleukin-17AT cellIn this issue of Immunity, Gomez-Rodriguez et al. (2009) demonstrate that signaling via the Itk kinase, a component of the T cell receptor signaling pathway, is required for interleukin-17A but not interleukin-17F expression in T helper 17 cells.
Martin et al. (2009)IL-17T cellsAhR expression in gammadelta T cells was essential for the production of IL-22 but not for optimal IL-17 production.
Milner et al. (2008)IL-17T cellsWe observed that ex vivo T cells from subjects with HIES failed to produce IL-17, but not IL-2, tumour-necrosis factor or interferon-gamma, on mitogenic stimulation with staphylococcal enterotoxin B or on antigenic stimulation with Candida albicans or streptokinase.
Suto et al. (2008)IL-17AT cellsUnexpectedly, we found that under Th17-polarizing conditions, the majority of IL-21-producing CD4(+) T cells did not produce IL-17A and -17F.