Viewing negative mentions of gene expression of Il12a (M. musculus) in T cells

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Kobayashi et al. (2002)IL-12T cellsIn contrast, IL-12 failed to produce any antitumor effect in athymic mice which lack conventional T cells.
Tamura et al. (2001)IL-12T cellThe same IL-12 EGT applied to the nude mouse model was not effective, suggesting the critical role of T cell infiltration in this treatment.
Caminschi et al. (1999)IL-12 transfectantT cellsThe IL-12 transfectant (AB1-IL-12) did not produce tumors in normal mice, but did so in athymic nude mice, implicating T cells in the prevention of MM tumor growth.
Guler et al. (1999)IL-12T cellsIn contrast, under the same conditions, BALB/c T cells fail to express IL-12 receptors and become unresponsive to IL-12, precluding any Th1-inducing effects if subsequently exposed to IL-12.
Gyülvészi et al. (2009)IL-12T cellsWe observed that T cells, which lack IL-12Rbeta1 (no IL-12 and IL-23 signaling), fail to invade the CNS and do not acquire a Th17 phenotype.
Moretto et al. (2010)interleukin-12T-cellIn the present study, we demonstrated that p40-deficient animals, which are unable to produce interleukin-12 (IL-12), have a serious defect in expansion of the CD8(+) T-cell response which compromises the survival of an infected host.
Eriksson et al. (2001)IL-12T cellsIn the absence of IL-12, autospecific CD4(+) T cells proliferated poorly and showed increased Th2 cytokine responses.
Jankovic et al. (2002)IL-12T cellIn the absence of IL-12, CD4(+) T cell responses to intracellular pathogens fail to default to a Th2 pattern and are host protective in an IL-10(-/-) setting.
Wüthrich et al. (2005)IL-12T cellDespite differential dependency of T cell subsets on IL-12 during vaccination, neither subset acquired memory immunity in the absence of IL-12.
Cousens et al. (1999)IL-12T cellAlthough interleukin (IL)-12 has the potential to contribute, IL-12-dependent T cell IFN-gamma has not been detected during viral infections.
Cong et al. (2000)IL-12BirThe two Bir T cell lines that did not induce lesions after transfer failed to significantly express CD40L or increase IL-12 when cultured with Ag-pulsed APCs.
Wykes et al. (2007)IL-12T cellsThese studies showed that DCs from lethal infections, previously shown to be unable to produce IL-12 (Figure 3) or present antigen to T cells (Figure 4), were unable to mediate survival from lethal disease.
Knolle et al. (1999)IL-12T cellsActivation of naive CD4(+) T cells by LSECs occurred in the absence of IL-12.
Madoiwa et al. (2009)IL-12T cellsThe CD4(+) T cells from thymic-injected mice could not proliferate or produce interleukin (IL)-2, IL-12 and IFN-gamma in response to FVIII.
Watford et al. (2008)IL-12RT cellsBecause resting T cells do not express IL-12R, PBMC from healthy adult donors (Department of Transfusion Medicine, Clinical Center, National Institutes of Health) were activated by culturing for 72 h in complete RPMI containing 1 ?
Jobe et al. (2009)IL-12T cells+ DC from livers of naďve challenged mice do not produce detectable levels of IL-12 and this also could have compromised their capacity to activate CD8+ T cells.
Byun et al. (2010)IL-12T lymphocytesGISF (50 and 150 kGy) augmented immune responsiveness via activation of NK cells, T lymphocytes proliferation, NO production, and cytokine level, such as IL-6, IL-2, IL-12, IFN-gamma, TNF-alpha, as compared with NISF, which strongly suggested that GISF significantly augmented an important element of all aspects of the innate and adaptive immune system.
Fransen et al. (2010)IL-12T cellInterestingly, it has been shown that DCs that had a mature phenotype but did not produce IL-12 and other pro-inflammatory cytokines, promoted expansion of CD4+ T cell populations lacking effector function [56].
Hamzeh-Cognasse et al. (2008)IL-12T cellFinally, we observed that while DCs in direct contact with PLTs neither exhibited an activated phenotype nor produced IL-12(p70), they could stimulate autologous T cell proliferation, although to a lesser extent than DCs cultured separately from PLTs (Figure 6B).
Woelbing et al. (2006)IL-12T cellsIn contrast, complement receptor 3-dependent phagocytosis of L. major by macrophages (MPhi) leads exclusively to MHC class II-restricted antigen presentation to primed, but not naive, T cells, and no IL-12 production.
Tanaka et al. (2007)IL-12T cellsIt was found that CD4+ T cells expressed the mRNA of 17 cytokines except for IL-12 and IL-15, while CD8+ T cells expressed only the mRNA of 9 cytokines after Con A stimulation.