Viewing negative mentions of gene expression of Fas (M. musculus) in T cells

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Document Target Regulator Anatomy Sentence
Andjeli? et al. (1994)FasT cellFas was not expressed on fetal nor adult CD8-CD4- (double-negative, DN) T cell precursors.
Van Parijs et al. (1996)FasT cellsUsing cells from TCR transgenic mice that do or do not express Fas, we show that there are two mechanistically distinct forms of apoptosis in CD4+ T cells.
Suarez-Pinzon et al. (1999)FasT-cellsIn contrast, Fas was expressed on CD4+ T-cells, CD8+ T-cells, and beta-cells in islet grafts from diabetic mice, but it was nearly or totally absent on these cells in islet grafts from normoglycemic mice.
Liu et al. (1999)FasT-cellSignificant lung injury developed in the transgenic CD8(+) T-cell recipients, whether or not Fas was expressed in these animals.
Arase et al. (1995)FasT cellsFas ligand mRNA was expressed in freshly isolated NK cells but not in T cells.
Kayser et al. (2008)FasT-cellDonor Fas is not necessary for T-cell-mediated rejection of mouse kidney allografts.
Chaouat and Clark (2001)LprT cellsLpr/lpr mice lack FAS and their T cells should remain effective at the fetomaternal interface.
Komano et al. (1999)lprT cellsThe homozygous lpr/lpr mice carrying the transgene did not produce anti-double-stranded DNA antibodies throughout their lives, whereas the development of abnormal lpr T cells (double negative, B220(+)) was not suppressed.
Van Houten and Budd (1992)lprT lymphocytesMRL-lpr/lpr (lpr) mice develop a polyclonal accumulation of abnormal peripheral T lymphocytes, which bear surface alpha beta TCR, CD3, and the B220 isoform of CD45, but lack CD4, CD8, and CD2.
Fortner and Budd (2005)FasT cellsIn the absence of Fas, T cells accumulate to significantly higher levels after transfer into lymphopenic recipients.
Hehner et al. (1999)CD95T-cellsT-cells lacking the CD95 receptor or caspase-8 and T-cells stably overexpressing a transdominant negative form of the adaptor protein FADD were still susceptible to pervanadate-induced apoptosis, excluding the involvement of the CD95 system or other FADD-dependent death receptors.
Dirsch et al. (2001)CD95T cellsWe show here that the sesquiterpene lactone helenalin (10-50 microM) induces apoptosis in leukemia Jurkat T cells even if they lack the CD95 death receptor or overexpress the antiapoptotic proteins Bcl-x(L) or Bcl-2.