Viewing negative mentions of gene expression of IL17A (H. sapiens) in T cells

Full-text article links are indicated by after the article reference.

Document Target Regulator Anatomy Sentence
Shin et al. (1999)IL-17T cellsIn contrast, naive CD45RA+T cells were unable to express IL-17 whatever the culture conditions.
Albanesi et al. (1999)IL-17T cellIn contrast, two nickel-specific CD8+ T cell clones failed to synthesize IL-17.
Gullick et al. (2010)IL-17 mRNAT cellsOther studies also report that IL-17+ T cells or IL-17 mRNA are not universally present in RA ST, and are found in 28–60% of RA ST samples [12], [23], [28].
Shah et al. (2010)IL-17T cellsAlthough the reason for this discrepancy is not clear, it could be related to the fact that this study noticed an increased frequency of DN T cells but not CD4+ T cells producing IL-17.
Chen et al. (2006)IL-17T cellsWe have shown that IL-23-dependent, pathogenic T cells produced IL-17 A, IL-17 F, IL-6, and TNF but not IFN-gamma or IL-4.
Res et al. (2010)IL-17AT cellThe origin of IL-22 single-producing T cells is unknown to date, but our results suggest that at least part of these cells may derive from Th17 and Tc17 cells, as we found that some of our IL-17A producing CD4 and CD8 T cell clones contained a proportion of cells producing IL-22 but lacking IL-17A and IFN-?.
Parsonage et al. (2008)IL-17T cellsEqually some CD4 T cells did not express IL-17 (Figure 1a,b).
Baba et al. (2010)IL-17T-cellWe found that CD45RA(+) Treg promoted while TNF-alpha inhibited naive CD4(+) T-cell differentiation into IL-17 and CCL20 co-expressing T(h)17 cells without influencing their IL-22 release.
Rao et al. (2008)IL-17T cells, and IL-21, have been suggested to contribute to the development of IL-17producing T cells in vitro (12, 31–35, 49), little information exists on which factors may be relevant in human immune responses in vivo (50).
Sender et al. (2010)IL-17T cellIL-23 propagates development of Th17 cells, a novel T cell subset which produces IL-17 but no interferon-gamma or IL-4.
Thorborn et al. (2010)IL-17T-cellIt is important to establish whether or not a significant reduction in the capacity of effector cells to produce IL-17 upon activation through the T-cell receptor complex can be validated in cells isolated ex vivo from blood using larger sample numbers than that used in this study and if a reduction of these effectors translates to overall reduction in IL-17 affecting Treg/Th17 balance or is compensated for by IL-17 production by other cell types.
Kim et al. (2005)IL-17T cellsIL-17 is produced mainly by activated CD4+ T cells, especially for Th1/Th0 cells, not the Th2 phenotype [26].
Pandolfi et al. (2009)IL-17AT-cells [97], it has been verified that the absence of IL-17A or IL-17R in T-cells led to an accelerated and severe wasting disease accompanied by higher expression of genes encoding TH1-type of cytokines.
Kuka et al. (2010)IL-17T cellsHowever no IL-17 was detected in naďve T cells differentiated in the presence of supernatants from PolyI?
Ma et al. (2008)IL-17T cellsHuman naive CD4+ T cells do not produce IL-17 when stimulated through CD3 and CD28 (11, 12).
Zhang et al. (2010)IL-17-secretingT cellsWe also found IL-17-secreting T clones highly expressing Foxp3, and with the suppression function to naďve T cells from NPC TIL in vitro (unpublished data), the failure to find the double IL-17 and Foxp3 positive TIL in NPC may be due to the lack of sensitivity of the IHC staining method or the differences between in vivo and in vitro conditions.
Ma et al. (2008)IL-17AT cellsIn complete contrast, when naive HIES CD4+ T cells were cultured under the same optimized conditions they failed to produce IL-17A (Fig. 4 A).
Ma et al. (2008)IL-17T cellBecause IL-17producing cells were absent from the CD4+ T cell compartment of HIES patients, we enumerated CD4+ CCR6+ T cells in peripheral blood.
Putheti et al. (2010)IL-17AT cellst, in in-vitro generated human CD4+IL-9+ T cells, although these cells do not express IL-17A or IL-17F expression.
Yao et al. (2010)IL-17T cellsTo address these questions, we isolated lymphocytes from BAL fluid, and right caudal lung lobe (infection site), spleen and mesentery lymph nodes obtained from the M. tuberculosis-infected macaques, and measured T cells actively producing IL-17 or IL-22 without in vitro antigen re-stimulation.
Yao et al. (2010)IL-17T cellsInterestingly, tuberculosis-driven T cells can actively produce IL-22 or IL-17 de novo without the requirement of in vitro antigen re-stimulation.
Lünemann et al. (2008)IL-17T cellIn contrast, IL-17 could not be detected in any EBNA1-stimulated T cells, neither in patients with MS nor in healthy virus carriers, indicating that deregulated T cell response to EBNA1 in MS is mediated by Th1, and not by Th17, cells.
Putheti et al. (2010)IL-17AT cellsAs analyzed by pre-amplification boosted single-cell real-time PCR, human CD4(+)IL-9(+) T cells expressed GATA3 and RORC, but not IL-10, IL-13, IFNgamma or IL-17A/F.