Viewing negative mentions of gene expression of IL6 (H. sapiens) in T cells

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Villiger et al. (1991)IL-6T cellAnalysis of purified T-alpha beta and T-gamma delta cells showed that neither T cell subset produced IL-6.
Villiger et al. (1991)IL-6T cellIn contrast, IL-6 was not detectable in non-infected T cell lines.
Gooding et al. (1993)IL-6T cellsThe cytokines IL-6 and IL-1 beta were detectable in the supernatant from whole marrow cultures and from cultures depleted of T cells but not in cultures depleted of mononuclear phagocytes.
Leenaerts et al. (1992)IL-6T cellsIrradiated or mitomycin-c treated PBMC could easily be induced (with LPS) to produce IL-6 and IL-1 while no activity was measured after 48 hr in the supernatant of PHA-stimulated T cells, suggesting that in the MLC the monokines were entirely produced by stimulator PBMC.
Vakkila et al. (1990)IL-6T cellsThus we concluded that IL-1 alpha, IL-1 beta, and IL-6 are primarily monocyte-derived factors and that these factors are not needed or produced during the activation of resting T cells by DC.
Mochizuki et al. (1996)IL-6T cellsThe following virologic, molecular biologic findings suggest that cytokines produced by HTLV-I-infected T cells in the eye play the central role in the pathogenic mechanisms of the uveitis: (a) the virus load in the peripheral blood monocytes analyzed by the quantitative polymerase chain reaction methods was significantly greater in patients with the uveitis than in asymptomatic carriers, (b) the proviral DNA of HTLV-I and the gene expression of the virus at the mRNA level was detected in the infiltrating cells from the eyes of the patients, (c) the virus particles were detected by electron-microscopic examination in the T-cell clones established from the intraocular fluid of the patients, and (d) the HTLV-I-infected T cells produced a variety of cytokines without any stimuli, such as interleukin (IL)-1 alpha, IL-2, IL-3, IL-6, IL-8, IL-10, tumor necrosis factor alpha, interferon-gamma, and granulocyte-macrophage colony-stimulating factor.
Fujihashi et al. (1996)IL-6T cellsCD4(+) T cells from diseased periodontal tissues are divisible into two groups based upon whether or not IL-10 is produced, together with IFN-gamma, IL-6 and IL-13.
Smith-Norowitz et al. (2008)IL-6T cellsCD8(+)CD60(+) T cells of the RS patients produced IL-2, IL-4, IL-10, IL-12, IFN-alpha. and IFN-gamma, but not IL-6 or IL-13.
Chen et al. (2006)IL-6T cellsWe have shown that IL-23-dependent, pathogenic T cells produced IL-17 A, IL-17 F, IL-6, and TNF but not IFN-gamma or IL-4.
Gruaz et al. (2010)IL-6T cellInterestingly, the cytokines induced by T cell contact in monocytes comprised IL-1beta, IL-6 but not IL-12, suggesting that this mechanism might favor Th17 polarization, which emphasizes the relevance of this mechanism to chronic inflammatory diseases and highlights the contrast with acute inflammatory conditions that usually involve lipopolysaccharides (LPS).
Lee et al. (1993)IL-6T-cellsPurified T-cells did not produce significant levels of TNF-alpha, even in the presence of IL-1 and IL-6.
Piuvezam et al. (1993)IL-6T cellT cell lines generated against Tct proliferated in response to parasite lysate only in the presence of autologous APC and produced IL-2, IL-6, and IFN-gamma but not IL-4 in response to PMA plus ionomycin.
Sissolak et al. (1992)IL-6T-cellBefore monocyte and T-cell depletion there was no expression of IL-1, IL-6 or GM-CSF, and only three of 13 patients studied expressed TNF mRNA.
Kitani et al. (1992)IL-6T cellsNormal T cells and B cells did not produce significant amounts of IL-6.
Ballow et al. (1996)IL-6T cellsAlthough interleukin (IL)-2, IL-4, and IL-6 could not be detected by ELISA in the T-cell-derived supernatants, RA probably generates a cytokine/interleukin from T cells which modulates B-cell Ig secretion.
Reid et al. (2005)IL-6T cellsCpG stimulation of ALL blasts produced increased levels of interleukin-6 (IL-6), IL-8, and IL-10 but no detectable IL-12p70 and led to a skewing of allogeneic T cells, with enhanced interferon gamma (IFN-gamma) production and reduced secretion of IL-5.
Nanki and E Lipsky (2000)IL-6T cellsIL-2, IL-4, and IL-6 were not expressed by the synovial tissue CD4+CD45RO+ T cells, whereas 2-20% of cells expressed the other cytokine mRNAs.
Nanki and E Lipsky (2000)IL-6T cellsIL-2, IL-4, and IL-6 were not expressed by the synovial tissue CD4+CD45RO+ T cells, whereas 2-20% of cells expressed the other mRNAs.