Viewing negative mentions of gene expression of IL2RA (H. sapiens) in T cells

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Yotnda et al. (1999)CD25T cellsIn bone marrow of ALL patients, T cells do not express CD40L and CD25 markers, their apoptosis rate is increased, and a predominance of a CD4 cell subset expressing a Th2 phenotype is detected.
Miescher et al. (1986)IL 2RT lymphocytesIn most TIL preparations, however, the T lymphocytes did not express the IL 2R and failed to proliferate in response to IL 2.
Smith and Roberts-Thomson (1990)IL2rT lymphocytesSynovial fluid T lymphocytes contain a subpopulation of larger cells expressing MHC class II and other lymphocyte activation antigens with the exception of the IL2r.
Morikawa et al. (1994)CD25T cellsAt concentrations of 1.6 to 40 micrograms/ml, these drugs suppressed interleukin-2 (IL-2) production induced by mitogen-stimulated T cells, but not the expression of IL-2 receptor (CD25), in a dose-dependent manner.
Pfoertner et al. (2006)CD25T cellsFurthermore, CD25 as well as other TReg cell molecules (for instance, GITR and CTLA4) are not expressed on all CD4+ T cells with regulatory function [15].
Masuda et al. (1994)CD25T cellPMT-2Y cells are positive for CD2, CD3, CD4, CD25, T cell receptor alpha beta and HLA-DR, but negative for CD1, CD7, CD8, CD19 and CD20, indicating that the clone belongs to a helper/inducer subset of T cells.
Elrefaei et al. (2009)CD25T cellspositive CD8+ T cells were FOXP3 negative and CD25 negative.
Trimble et al. (2000)CD25T cellsHowever, CD3zeta-CD28-T cells generally do not express CD25 after anti-CD3 and anti-CD28 stimulation and are not cytotoxic until they are cultured with IL-2 overnight.
Takami et al. (1998)CD25T-cellThe leukaemic cells were also positive for T-cell antigens such as CD2, CD3, CD5, CD7, CD8 and T-cell receptor (TCR) Vbeta8 and for activation antigens such as CD38 and HLA-DR, but were negative for CD19, CD21, CD22, CD25.
Matsuzaki et al. (1992)IL2-RT cellKHM-3S cells were positive for IL2-R (Tac) and NKH-1, but negative for other lymphocytic markers such as OKT 11, OKT 4, OKT 8, T cell receptor (WT 31), B 1, and B 4.
Junghans et al. (1993)interleukin 2 receptorT-cellsThe scientific basis for the clinical use of this antibody in radioimmunotherapy is that resting normal cells do not express the interleukin 2 receptor, whereas the receptor is expressed on the surface of certain neoplasms and by activated T-cells in select autoimmune diseases and in allograft rejection.
Yodoi et al. (1987)IL-2-RT cellHTLV-I(+) leukemic T cells and T cell lines from patients with adult T cell leukemia (ATL) continuously expressed IL-2-R without production of IL-2.
Holán and Minowada (1993)IL-2RT cellsThe majority of IL-2 producing cell lines originated from less mature, non-activated T cells, as they were characterized by the expression of TdT, lack of HLA-DR antigens and > 50% had no detectable IL-2R alpha.
Konishi (1989)IL-2RT-cellsNorthern analysis using a 32P-labeled cDNA probe for the IL-2R alpha chain demonstrated that blood T-cells from patients with active sarcoidosis, but not from normal subjects express 3.5 kb and 1.5 kb IL-2R mRNA transcripts, identical to the observation when normal T-cells are activated in vitro.
Yamada et al. (1987)IL 2RT cellsWe were unable to detect interleukin 2 receptors (IL 2R) on fresh LGL and T cells using flow cytometric analysis with anti-Tac monoclonal antibody or radiolabeled probes with [125I]anti-Tac.
Bujdoso et al. (1993)IL-2RT cellsAll gamma/delta T cells express IL-2R within 12 h following activation by Con A in vitro whereas alpha/beta T cells do not express the IL-2R until 24 h after the start of culture with the mitogen.
Aune and Pogue (1989)IL-2RT cellsCD4+ T cells stimulated with PWM produced equivalent amounts of IL-2 in the presence or absence of Ts cells but failed to express the IL-2R (TAC) on their surface in the presence of Ts cells.