Viewing negative mentions of gene expression of IGHE (H. sapiens) in T cells

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Gauchat et al. (1991)IgET-cellHere we show that highly purified B-cells (greater than 99.5%) failed to produce IgE in the presence of IL-4 but IgE synthesis was restored by the addition of autologous CD4+ T-cells or by CD4+ T-cell clones with non-relevant specificities, derived from different donors.
Uchiyama et al. (1980)IgCTCThe CTC did not produce significant amounts of Ig in the presence of any polyclonal activator.
Matsuo et al. (2000)IgT-cellThe immunoprofile of MOLP-5 corresponds to that seen typically in primary MM cells: positive for cytoplasmic immunoglobulin (Ig) kappa light chain, CD28, CD29, CD38, CD40, CD44, CD49d, CD54, CD56, CD58, CD71, CD138 and PCA-1; the cells were negative for surface Ig and various other B-cell, T-cell and myelomonocyte-associated immunomarkers.
Saxon et al. (1992)IgET-cellHowever, we found that the patients' cells do not make IgE in response to an IL-6/T-cell-dependent differentiation pathway employing exogenous interleukin-4 (IL-4).
Shiobara et al. (1984)IgT cellsCo-culture of T and non-T cells from 10 of 12 patients failed to produce Ig.
Matutes et al. (1987)IgT-cellThe leukaemic cells resembled small, mature lymphocytes but lacked B- and T-cell membrane markers as well as immunoglobulin (Ig) and T-cell receptor gene rearrangements.
Tao and Kahn (2000)IgT-cellImmunophenotyping of the lung and lymph node lesions revealed identical surface marker profiles: cells were CD19(+), CD20(+), immunoglobulin (Ig) G(+), kappa(+), lambda(-), CD5(-), CD10(-), CD23(-), and IgM(-), and also negative for T-cell markers.