Viewing negative mentions of gene expression of HLA-DRA (H. sapiens) in T cells

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Tejani et al. (1999)HLA-DRT-cellFactors predictive of DGF in CD patients were: African-American race (25%), prolonged cold ischemia (24%), absence of T-cell induction antibody therapy and absence of HLA-DR matching.
Reitz et al. (1984)DR alpha mRNAT-cellsIn DNA from the T-cells not expressing DR alpha mRNA, all of the potential HpaII sites within the BglII fragment appeared to be methylated.
Tabibzadeh (1990)HLA-DRT-cellsInterleukin-2 receptor was not expressed in human endometria; however, endometrial T-cells within lymphoid aggregates, some scattered in endometrial stroma and a few within epithelium, expressed HLA-DR, HLA-DP, HLA-DQ, and VLA-1.
Testa et al. (2002)HLA-DRT-cellThe blasts were CD34(+), AC133(+), CD71(-), HLA-DR(-), CD38(-/dim+), CD90(+), CD117(dim+), flt3(+); did not express B, T, or myeloid-associated antigens; and showed a germline configuration of the immunoglobulin and T-cell receptor.
Shikata et al. (2009)HLA DRT cellSeveral additional immunohistochemical staining showed that the cells were positive for CD13, CD34, CD117, and HLA DR, but negative for myeloperoxidase, CD42b, glycophorin, B cell marker, T cell marker, cytokeratin and desmin.
Ghosh et al. (1985)HLA-DRT cellsThe majority of lymphoid cells in reactive effusions were T cells, which lacked HLA-DR and showed a marked excess of helper/inducer cells (mean helper to suppressor ratio of 3 X 5).
Lamour et al. (1995)HLA-DRT cellsThe majority of Fc gamma RIII-positive gamma delta T cells do not express HLA-DR in patients with primary Sjögren's syndrome.
Gebel et al. (1987)HLA-DRT cellsAlthough resting T cells from normal subjects did not express HLA-DR or HLA-DQ molecules, up to 86% (range 22-86%) of T cells from BMT recipients expressed HLA-DR.
Ashton-Key et al. (1996)HLA-DRT cellCONCLUSIONS: These data show that low or absent HLA-ABC and HLA-DR antigen expression occurs commonly in enteropathy associated T cell lymphoma.
Vermijlen et al. (2010)HLA-DRT cellsT cells from CMV-infected newborns expressed the activation marker HLA-DR, whereas expression was virtually absent in uninfected controls (Table I).
Bagby et al. (1981)HLA-DRT lymphocytesThe T lymphocytes that interact with monocytes and lactoferrin to inhibit CSA production are similar to those that augment CSA production because their activities are neither genetically restricted not glucocorticoid sensitive, and both populations express HLA-DR (Ia-like) and T3 antigens but not T4 or T8 antigens.
Wieczorek et al. (1986)HLA-DRT-cellIn each case the large, pleomorphic neoplastic cells lacked the monoclonal antibody-defined cell surface antigens commonly associated with immature and mature T-cells, i.e., T11, Leu-1, T3, T4, T6, T8, and T10, but expressed various T-cell-associated activation antigens, such as HLA-DR, Tac, and T21.
Stela et al. (1990)HLA-DRT-cellThe E receptors, the T-cell antigens, the HLA-DR antigens and smIgG were either expressed or not according to the affected organ, the progress of illness, or the treatment.
Vanham et al. (1993)HLA-DRT-cellRESULTS: No effect was observed on CD4 and CD8+ T-cell counts or on CD8+ T-cell activation markers, except for a selective increase in HLA-DR expressing CD8 cells in the DTC-treated group.
Porakishvili et al. (2001)HLA-DRT cellsIn contrast, this population lacked expression of either CD69 or HLA-DR, arguing that they were not activated or that they are an abnormal population of T cells.