Viewing negative mentions of gene expression of CTLA4 (H. sapiens) in T cells

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Liu et al. (1998)CTLA-4T cellsBut patients with SLE had significantly increased percentages of CTLA-4-positive T cells compared with normal controls, implying at least that there was no apparent defective expression of CTLA-4 molecule in human lupus.
Schlotmann et al. (2000)CTLA-4T cellsAfter initiation of antiplasmodial treatment, it returned to control values within a few days. gammadelta T cells, which also are implicated in the pathogenesis of human malaria, did not express CTLA-4.
Xerri et al. (1997)CTLA4T-cellCTLA4 was present in neoplastic cells from most (10/11) T-cell malignancies, except for anaplastic and lymphoblastic subtypes (0/4).
Newland et al. (2004)CTLA4T cellCTLA4 (CD152) is a transmembrane molecule expressed on activated T cells and functions as a negative regulator of T cell activation upon binding to the costimulatory molecules CD80/86.
Perkins et al. (1996)CTLA4T cellThe CTLA4 molecule is not expressed on resting T cells, but is induced after the initial steps of T cell activation.
Boulougouris et al. (1998)CTLA-4T cellsIn the absence of intracellular calcium elevation, CTLA-4 was not expressed at the cell surface, and CD80 acted positively as a costimulator of T cells, via CD28.
Kuiper et al. (1995)CTLA-4T cellsPurified B cells did not express CTLA-4 when mitogenically activated with alpha IgM and CD40 Ab, but did express the molecule when cultured in the presence of membranes from activated T cells, which suggests that induction of CTLA-4 expression on B cells was dependent on direct cell-cell contact of B lymphocytes and activated T cells.
Lozanoska-Ochser et al. (2008)CTLA-4IgT cellIdentical results were obtained for T cell adhesion using either CTLA-4 blocking mAb or CTLA-4Ig (abatacept), indicating CTLA-4 as the T cell ligand for these CD86-mediated effects.
Freeman et al. (1992)CTLA-4T cellsCD28 was detected in resting T cells, whereas CTLA-4 was not.
Hoffmann et al. (2006)CTLA-4T-cellFurther analysis revealed that these cells originate from CD45RA+ naive cells within the CD4+ CD25high T-cell compartment, as only this subpopulation homogeneously expressed CD62L, CCR7, cytotoxic T lymphocyte-associated antigen-4 (CTLA-4), and forkhead box P3 (FOXP3), produced no inflammatory cytokines and maintained robust suppressive activity after expansion.
Pfoertner et al. (2006)CTLA4T cellsFurthermore, CD25 as well as other TReg cell molecules (for instance, GITR and CTLA4) are not expressed on all CD4+ T cells with regulatory function [15].
Zaunders et al. (2006)CTLA-4T cellsThe majority of these initial vaccinia virus-specific CD4+ T cells were CD127+ and produced interleukin-2 (IL-2) but not CTLA-4 in response to restimulation in vitro.
Elrefaei et al. (2009)CTLA-4T cellsand IL-10 positive CD8+ T cells did not express CTLA-4.
Joosten et al. (2010)CTLA4T-cellsAs mentioned above, the HLA-E/peptide specific T-cells also expressed several markers that have been associated with human CD8+ Tregs, notably CD25 and LAG-3 in the absence of CD127 [32], although they did not express FoxP3, GITR and CTLA4 (Figure 3).
Damle et al. (1994)CTLA-4T cellsResting CD4+ T cells express CD28 but not CTLA-4 on their surface.
Elrefaei et al. (2009)CTLA-4T cellsTGF-beta and IL-10 positive CD8+ T cells did not express CTLA-4.
Elrefaei et al. (2010)CTLA-4T cellsInterestingly, HIV-specific TGF-beta-positive CD4(+) T cells did not substantially express CTLA-4.
Kato et al. (2001)CTLA-4T cellsThe CD28(-)CD4(+) T cells did not express detectable levels of CD25, CD69, V(alpha)24, or CTLA-4 but expressed heterogeneous amounts of CD45 RA on the surface.
Wan et al. (2006)CTLA-4T cellsIn this study, initial analysis of costimulatory molecules led to the unexpected observation that, in addition to CD80, several negative regulators (e.g., CTLA-4, programmed death-1 (PD-1), and PD ligand-1) were overexpressed in synovial T cells and macrophages derived from RA patients as opposed to controls.
Jaffar et al. (1999)CTLA-4T cellsFACS analysis revealed that stimulated asthmatic bronchial tissue was comprised of CD4+ T cells that expressed surface CD28 (75. 3%) but little CTLA-4 (4.0%).
Ahmadzadeh et al. (2009)CTLA-4T cellsPD-1(+) TIL expressed CTLA-4 and Ki-67, markers that were not expressed by PD-1(-) TIL and T cells in the normal tissues and PBL.