Viewing negative mentions of positive regulation of CEL (H. sapiens) in T cells

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Beyersdorf et al. (2005)cellT cellThe molecular properties of superagonistic anti-CD28 antibodies allow the generation of a strong activating signal in mature T cells, including T(reg) cells, without additional stimulation of the T cell receptor complex.
Watanabe et al. (1998)cellT cellFlow cytometric analysis demonstrated that CD4+ intraepithelial T cells were increased, but T cell receptor gamma/delta T cells and CD8alpha/alpha cells were not increased in the area of chronic inflammation.
Samstag et al. (1988)cellT cellsOur results also show that substantial modulation of the T-cell receptor complex and most likely CD3 phosphorylation are not essential for initiating the activation of resting T cells.
Ariumi and Trono (2006)cellT-cellNotably, ATM overexpression did not stimulate the HIV-1 late gene expression within the context of Rev-independent constructs or the Rex-dependent production of capsid from human T-cell leukemia virus type 1 proviral constructs.
Yu et al. (2009)cellT cellsHere, we showed that Bcl-6 directs Tfh cell differentiation: Bcl-6-deficient T cells failed to develop into Tfh cells and could not sustain germinal center responses, whereas forced expression of Bcl-6 in CD4(+) T cells promoted expression of the hallmark Tfh cell molecules CXCR5, CXCR4, and PD-1.
Peavy et al. (2005)NFATNFATGqalpha and G14alpha stimulate nuclear factor of activated T cell (NFAT) activation in VSMC, but Galpha-induced cell death seems independent of PKC, InsP(3)/Ca(2+), and NFAT, in that pharmacological inhibitors of these pathways did not block cell death.
Shapiro et al. (2003)cellT-cellIn both cases the malignant cells were CD56+, CD2+, and terminal deoxynucleotidyl transferase (TdT) positive with no detectable T-cell receptor (TCR) gamma chain gene rearrangement.
Hsu et al. (1998)cellT-cellFinally, we show that a Wnt-1 signal and beta-catenin association are not required for the architectural function of LEF-1 in the regulation of the T-cell receptor alpha enhancer, which involves association of LEF-1 with a different cofactor, ALY.
Kuefer et al. (1997)cellT-cellGenotypic analysis showed that the tumors were B-lineage and clonal, with rearrangements of the Ig heavy- and kappa light-chain loci and no rearrangements of the T-cell receptor beta locus.
Breithaupt et al. (1993)cellT cellAnti-CD2-induced tyrosine phosphorylation of T cell polypeptides is independent of the PMA-induced modification of p56lck.
Murison et al. (1993)cellT cellWe observed that double negative TCR alpha beta+ T cells differ from single positive T cells in the following respects: (i) their T cell receptor (TCR) repertoire is different, as revealed by the analysis of 47 clones derived from three individuals and by analysis of peripheral blood lymphocytes (PBL) prior to in vitro manipulation; (ii) their in vivo CD3:TCR expression is lower before in vitro manipulation and expansion; (iii) their direct proliferative response to IL-3, which is not mediated by secondary release of other T cell growth factors.
Morishima et al. (1987)cellT cellPhorbol ester induces interleukin-2 receptor on the cell surface of precursor thymocyte leukemia with no rearrangement of T cell receptor beta and gamma genes.
Koizumi et al. (1986)cellT cellThe LGLs showed no rearrangement of T cell receptor C beta genes.
Wang et al. (2004)cellT cellThere was no significant difference of activated T cell CD3+/HLA-DR+ among patients of stage I,II,III.
Yablonski et al. (1998)cellT-cellA Nck-Pak1 signaling module is required for T-cell receptor-mediated activation of NFAT, but not of JNK.
Mutalithas et al. (2010)BALT cellsWithin paired blood–BAL samples from the same subject there was no increase in CRTH2+ T cells in the BAL compared to blood in asthmatics.
Smith (2006)cellT-cellIn contrast, those nef alleles that did not reduce cell surface expression of TCR-CD3 were found to increase T-cell activation and apoptosis.
Gu et al. (2007)cellT cellCulturing cells in the presence of IL-4 failed to enhance gag specific T cell responses.
Wang et al. (2010)cellT cellAs shown in Table 4, the anti-HLA-I antibody in fact showed partially blocking of reactivity for some peptide epitopes (PF106 and PF141) that, according to the cell depletion experiments (Fig. 1) induced CD4+ T cell, but not CD8+ T cell, responses.