Viewing affirmative mentions of gene expression of IL2 (H. sapiens) in CSF

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Richartz et al. (2005)IL-2CSFWe measured the concentrations of the pro-inflammatory cytokines IL-1beta, IL-2, IL-6, and TNF-alpha, and of the soluble receptors sIL-2r, sIL-6r, and sTNF-alphar, in cerebrospinal fluid (CSF) and serum of 20 Alzheimer patients and 21 controls.
Boutin et al. (1987)interleukin-2cerebrospinal fluidIn addition, soluble interleukin-2 receptors were detected in the cerebrospinal fluid from patients with acute primary encephalitis.
Sharief et al. (1992)IL-2CSFCSF IL-2/albumin ratios higher than concomitant serum ratios were considered indicative of intrathecal IL-2 production.
Ciardi et al. (1993)IL-2CSFLevels of IL-2 and soluble IL-2 receptor (sIL-2R) in serum and cerebrospinal fluid (CSF) samples from 31 asymptomatic HIV-1 seropositive individuals were measured and correlated with levels of anti-1 IgG and IgM antibodies.
Ciardi et al. (1993)IL-2CSFSimilarly, IgG antibodies were detected in 10 subjects who had elevated IL-2 levels in the CSF.
Gallo et al. (1989)IL-2cerebrospinal fluidSerum and cerebrospinal fluid (CSF) levels of interleukin-2 (IL-2), soluble IL-2 receptor (sIL-2R), and gamma-interferon (gamma-IFN) were measured in multiple sclerosis (MS) patients, human immunodeficiency virus type 1 (HIV-1)-infected patients and normal controls (NC).
Rapaport et al. (1997)IL-2CSFThere was a significant positive correlation between CSF IL-1 alpha and IL-2 levels for all subjects (r = 0.50, n = 44, p = 0.0001).
Ciardi et al. (1993)IL-2CSFHigh IL-2 levels were detected in the CSF of 20 (65%) subjects, 18 (90%) of whom had evidence of intrathecal synthesis of HIV-1-specific IgM antibodies.
Gallo et al. (1988)Interleukin-2cerebrospinal fluidInterleukin-2 levels in serum and cerebrospinal fluid of multiple sclerosis patients.
Gallo et al. (1988)interleukin-2cerebrospinal fluidA sensitive enzyme-linked immunosorbent assay method was employed to measure interleukin-2 (IL-2) levels in cerebrospinal fluid (CSF) and sera from 30 patients with multiple sclerosis (MS) and 8 patients with other neurological diseases.
Santoli et al. (1984)IL 2CSFConsistent with these data, the CSF-derived cultures displayed a variety of immunoregulatory functions, such as the ability to lyse nonspecific and PHA-stimulated target cells, to produce IL 2 upon mitogenic activation, and to modulate polyclonally induced Ig responses.
Sharief et al. (1991)interleukin-2cerebrospinal fluidThe patients with post-polio syndrome had significantly higher mean (+/- SD) (cerebrospinal fluid levels of interleukin-2 and soluble interleukin-2 receptors than the controls (8.1 +/- 5.3 vs. 1.4 +/- 0.8 U per milliliter and 159.6 +/- 102.9 vs. 10.7 +/- 6.2 U per milliliter, respectively).
Sharief et al. (1991)IL-2cerebrospinal fluidHigh cerebrospinal fluid levels of IL-2 and sIL-2R were detected mainly in patients with acute relapsing-remitting MS and were significantly higher than corresponding serum levels.
Sivieri et al. (1998)IL-2CSFIncreased serum and/or CSF levels of IL-2 and sIL-2R strongly confirm a CD4+ Th1 activation.
Sharief et al. (1993)IL-2cerebrospinal fluidTo evaluate the in vivo relationship of the IL-2 system to blood-brain barrier impairment in MS, levels of IL-2 and soluble IL-2 receptors (sIL-2R) in cerebrospinal fluid (CSF) and serum samples from 50 patients with active MS and 49 controls were correlated with values of the CSF to serum albumin ratio.
Gallo et al. (1989)interleukin-2CSFIn none of the CSF samples tumor necrosis factor-alpha or interleukin-2 was detected.
Ott et al. (1993)IL-2CSFCerebrospinal fluid (CSF) and serum levels of interleukin-2 (IL-2), soluble IL-2 receptors (sIL-2R), neopterin, L-tryptophan (L-TRP) and beta 2-microglobulin (beta 2-M) were measured in 31 untreated multiple sclerosis patients in acute exacerbation and 27 normal controls.
Shaw et al. (1995)interleukin-2cerebrospinal fluidConcentrations of neopterin, interleukin-2 (IL-2), soluble interleukin-2 receptor (sIL-2R) and tumour necrosis factor-alpha (TNF-alpha) were measured in the serum and cerebrospinal fluid of 19 clinically-inactive MS patients and compared with those of 19 non-inflammatory controls.
Ciardi et al. (1994)IL-2CSFLevels of TNF-alpha, IL-2 and soluble IL-2 receptor (sIL-2R) in serum and cerebrospinal fluid (CSF) samples from 31 asymptomatic HIV-1 seropositive individuals were measured.
Ciardi et al. (1994)IL-2CSFMoreover, CSF levels of TNF-alpha significantly correlated with CSF levels of IL-2 and sIL-2R.
Babu et al. (2006)IL-2CSFMETHODS: We measured the levels of TNF-alpha and IL-2 in the serum and cerebrospinal fluid (CSF) of patients suffering JE, using enzyme-linked immunosorbent assay (ELISA).
Brod et al. (1991)IL-2CSFWe found that most clones from both the peripheral blood and CSF express IL-1, IL-2, IL-4, IFN-gamma, or TNF-alpha cytokine mRNA after activation with ionomycin and PMA.
Ciardi et al. (1994)IL-2CSFHigh levels of TNF-alpha were detected in CSF of 17 (55%) and serum of 22 (71%) subjects, 15 (88%) of whom had elevated CSF IL-2 levels and 16 (94%) had high sIL-2R levels.
Gallo et al. (1991)interleukin-2cerebrospinal fluidThe cerebrospinal fluid (CSF) and serum levels of interleukin-2 (IL-2), soluble IL-2 receptor (sIL-2R), interleukin-4 (IL-4) and interferon-gamma (IFN-gamma) were longitudinally investigated in 20 multiple sclerosis (MS) patients.
Blum-Degen et al. (1995)IL-2cerebrospinal fluidInterleukin-1 beta (IL-1 beta), interleukin-2 (IL-2), and interleukin-6 (IL-6) were measured in the cerebrospinal fluid (CSF) and plasma of 12 control subjects, 11 sporadic Alzheimer's disease (AD) and 22 de novo Parkinson's disease (PD) patients using high sensitivity enzyme-linked immunosorbent assays (ELISA).
Babu et al. (2006)IL-2CSFRESULTS: JE infection caused a remarkable increase (p<0.0001) in the levels of TNF-alpha in the serum and CSF, while IL-2 levels were unaffected.
Meyer-Rienecker et al. (1979)lymphokineCSFMoreover, a characteristic lymphokine pattern for MS with high activities in all regions of molecular weight, especially in the CSF, could be detected.
Gilad et al. (1997)interleukin-2CSFCerebrospinal fluid (CSF) and serum samples of 20 young adults (mean age 41 +/- 3.4 yr) with a first episode of stroke were tested for interleukin-2 (IL-2), soluble interleukin-2 receptor (SIL-2R), tumour necrosis factor-alpha (TNF-alpha) and interleukin-1-beta (IL-1 beta) levels.
Warnke et al. (2010)Interleukin-2cerebrospinal fluidAlthough CD19+ B-cell reduction occurs rapidly, recovery from the nadir is seen earlier and more pronounced compared with T cells.56–60 Recent evidence indicates that cladribine may also impede the influx of T cells into the CNS, and might also influence levels of soluble adhesion molecular levels such as sICAM or sE-Selectin.61,62 In addition, cladribine may exert immunomodulatory effects on proinflammatory cytokine profiles: Mean values of Interleukin-2 (IL-2) and soluble interleukin-2 receptor levels measured 12 months after cladribine treatment for chronic progressive MS were found to be lowered.63 IL-8-levels were decreased in cerebrospinal fluid (CSF) of cladribine-treated RRMS patients, whereas CCL-5 levels were decreased both in CSF and serum.64 These and other data suggests that cladribine not only has an leukocyte depleting effect, but also may exert a direct effect on effectors T-cell function.59,65 Pharmacokinetics of cladribine
Buttinelli et al. (1992)interleukin-2cerebrospinal fluidT-cell clones derived from the CSF (cerebrospinal fluid) of MS (multiple sclerosis) patients have been analysed for the production of interferon-gamma (IFN-gamma), tumour necrosis factor-alpha (TNF-alpha), interleukin-2 (IL-2) and interleukin-4 (IL-4).
Mogi et al. (1996)IL-2cerebrospinal fluidInterleukin (IL)-1 beta, IL-2, IL-4, IL-6 and transforming growth factor-alpha levels are elevated in ventricular cerebrospinal fluid in juvenile parkinsonism and Parkinson's disease.
Mogi et al. (1996)IL-2VCSFIn contrast, the concentrations of IL-2 and IL-6 in VCSF from patients with PD were higher than those from control patients (P < 0.05).
Weller et al. (1992)IL-2CSFWe have analysed the natural immune response to leptomeningeal tumor invasion in 43 Patients by assessing cerebrospinal fluid (CSF) levels of albumin, IgG, IgM, interleukins (IL) 1, 2, 4 and 6, soluble IL-2 receptor (sIL-2R), interferon gamma (IFN gamma), tumor necrosis factor alpha (TNF alpha), and the tumor markers, carcinoembryonic antigen (CEA) and alphafetoprotein (AFP).
Tekgül et al. (1999)IL-2Cerebrospinal fluidCerebrospinal fluid and plasma TNF-alpha and IL-2 levels were nondetectable in two patients who had a stationary course of disease and were markedly elevated in patient 3, who displayed a rapidly progressive course.