Viewing negative mentions of phosphorylation of IL2 (H. sapiens)

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Document Target Regulator Anatomy Sentence
Gallis et al. (1986)IL-2The IL-2 receptor expressed by these cells was not phosphorylated in the presence of PMA.
Decker et al. (2010)IL-2Addition of IL-2 to activated B-CLL cells caused rapid phosphorylation of Akt, while IL-2 failed to significantly phosphorylate Akt in resting B-CLL cells.
Zu et al. (1990)IL 2After cell conversion from an IL 2 dependent state to an IL 2 independent state, one of the five phosphoproteins, the 65-kDa protein, became constitutively phosphorylated even without addition of IL 2.
Candotti et al. (1997)IL-2-mediatedAccordingly, IL-2-mediated phosphorylation of STAT5 was also absent or barely detectable.
Mills et al. (1992)IL-2Despite the absence of LCK or FYN, IL-2 induced similar patterns of rapid tyrosine phosphorylation.
Gilmour et al. (2001)IL-2-inducedNine had no detectable gammac, four had abnormal gammac expression and no IL-2-induced JAK3 tyrosine phosphorylation, and one had normal gammac expression but no IL-2-induced JAK3 tyrosine phosphorylation, although JAK3 was present.
Jensen et al. (2001)IL-2APC pulsed with S(72)(Tn), which differs from T(72)(Tn) by not having a methyl group in the serine amino acid side chain to which the glycan is attached, induced partial tyrosine phosphorylation of the TCR-zeta 21-kDa protein, no tyrosine phosphorylation of the MAP kinases and no IL-2 production.