Viewing negative mentions of negative regulation of IL2 (H. sapiens) in mononuclear cells

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Usui (1990)IL-2mononuclear cellsThe production of IL-2 by peripheral blood mononuclear cells (PBM) from patients with renal cell carcinoma was not suppressed, when compared with that in controls.
Reed et al. (1986)IL2PBMCAt concentrations that produced 75-90% suppression of proliferation in PHA-stimulated PBMC cultures, IDS caused no decrease in interleukin 2 (IL2) production (determined by bioassay) or in IL2 receptor expression (determined with anti-Tac antibody).
Burns et al. (1996)IL-2PBMCIL-2 production by PBMC in response to Daudi cells was also decreased by depletion of CD4+ T cells, but not by depletion of CD8+ or gammadelta T cells.
Mantovani et al. (1988)IL-2PBMCWe found no significant defect in the IL-2 receptor expression by PBMC of cancer patients.
Linker-Israeli and Casteel (1988)IL-2PBMSF PBM suppressed production of IL-2, but not proliferative responses to this lymphokine.
Reed et al. (1985)IL 2PBMCOur data revealed that although PBMC-proliferation was markedly suppressed by WGA, levels of IL 2 activity in WGA-inhibited cultures were not reduced, but instead were increased, suggesting failure to utilize IL 2.
Ampel and Kramer (2003)IL-2mononuclear cellsDepletion of NK cells from peripheral blood mononuclear cells resulted in significant increases in TNF-alpha and IL-10 (for both, p<0.05) but resulted in no significant decrease in IFN-gamma or IL-2.
Dooper et al. (2002)IL-2PBMCWe found that tumour necrosis factor-alpha (TNF-alpha), interferon-gamma (IFN-gamma) and to a lesser extent interleukin (IL)-10 production was inhibited by all PGE-subtypes in ConA-stimulated PBMC concomitant with unaffected IL-2 levels.
Sironi et al. (1997)lymphokinemononuclear cellsUnlike benzydamine, ibuprofen and naproxen, two non-steroidal antiinflammatory drugs also used topically, were unable to suppress inflammatory lymphokine production from Candida-activated mononuclear cells.