Viewing negative mentions of gene expression of IFNG (B. taurus) in T cells

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Bassey and Collins (1997)IFN-gammaT cellsSimilar responses to A-PPD were observed except that PBMC produced higher levels of IFN-gamma than did CD4+ T cells.
Hodge et al. (2009)IFN-gammaT-cellT-cell IFN-gamma and TNF-alpha was unchanged in the blood of BOS patients compared with healthy controls.
Shi et al. (2008)IFN-gammaT cellsIn the absence of Jak3-dependent signals, naive CD4+ T cells proliferate robustly but produce little IFN-gamma after Th1 cell polarization in vitro.
Simutis et al. (2007)IFN-gammaT cellsTaken together, the data suggest that (1) gammadelta T cells do not produce significant IFN-gamma and do not significantly increase NO production from M. paratuberculosis-infected macrophages in vitro, (2) the production of significant IFN-gamma by antigen-stimulated CD4+ T cells from infected calves is insufficient to enhance mycobacterial killing or nitrite production by infected macrophages, and (3) macrophages may have an impaired NO response following intracellular M. paratuberculosis infection, even in the presence of significant concentrations of IFN-gamma.
Fikri et al. (2000)IFN-gammaT cellsWe found that purified uncultured WC1+ T cells express TNF-alpha, CD28, CTLA-4 and IL-2R alpha mRNA transcripts but do not express those for IL-2, IL-4, IL-6, IL-10 and IFN-gamma.
Brown et al. (1994)IFN-gammaT cellsThe gamma/delta T cells were IL-2 dependent and expressed IFN-gamma and TNF-alpha, but not TNF-beta, IL-2, or IL-4 mRNA.
Till et al. (1997)IFN-gammaT cellsIL-13 production by allergen-stimulated T cells is increased in allergic disease and associated with IL-5 but not IFN-gamma expression.
Brugnolo et al. (1999)IFN-gammaT cellsStreptokinase-specific T cells from all subjects showed intracellular expression of IFN-gamma with poor or no IL-4, whereas Der p 1-specific T cells exhibited IFN-gamma but low or no IL-4 expression in nonatopics, and remarkable IL-4 expression in atopic donors.
Brugnolo et al. (1999)IFN-gammaT cellBy contrast, all penicillin G-, ampicillin-, and amoxicillin-specific short-term T cell lines showed high intracellular expression of IL-4, IL-5, and IL-13, but poor or no expression of IFN-gamma, thus exhibiting a clear-cut Th2 profile.
Jirapongsananuruk and Leung (1997)IFN-gammaT cellsThe majority of T cells in early allergic reactions are T helper type 2 (TH2)-like producing IL-4, IL-5, IL-13 but not IFN-gamma.
Ovigne et al. (2001)interferon-gammaT cellsThe majority of epidermal CD8+ T cells in chronic plaque psoriasis are activated Tc1 cells producing interferon-gamma and no interleukin-4, a small proportion of which express NK-T receptors.
Ovigne et al. (2001)interferon-gammaT-cellAll the TCL and 37/39 CD8+ T-cell clones produced interferon-gamma but no or minimal interleukin-4 or interleukin-10.
Williams et al. (2007)IFN-gammaT cellsFinally, we show that CD25-deficient CD4 T cells produce little IFN-gamma in the presence of OX40 costimulation compared with wild type, suggesting that IL-2R signaling is required for efficient OX40-mediated differentiation to IFN-gamma secretion.
Brown et al. (2000)interferon-gammaT cellIn contrast, CD4+ T cells in five of 12 T cell lines obtained from disease controls did not produce or produced minimal interferon-gamma in response to group A streptococcal isolates; this was significantly different from the psoriatic T cell lines (p < 0.05).
Satoguina et al. (2002)IFN-gammaT cellsHere we show that in the chronic helminth infection onchocerciasis (river blindness), where patients have relatively little sign of dermatitis despite the presence of millions of small worms in the skin, T cells can be obtained which bear characteristics of Tr1 cells, producing no IL-2 or IL-4 but substantial amounts of IL-10, variable amounts of IL-5, and some IFN-gamma.
Aso et al. (2006)IFN-gammaT lymphocytesWe found no significant difference in IFN-gamma-producing T lymphocytes between healthy controls and patients with only type 1 diabetes (n = 8) or Graves' disease (n = 5).
Erard et al. (1999)IFN-gammaT cellsIndeed, while presence of TGF-beta biases the development of CD8 T cells that, then, produce little cytolytic activity and IFN-gamma, addition of IL-4 results in the recovery of cytotoxicity and IFN-gamma production.
Milner et al. (2008)interferon-gammaT cellsWe observed that ex vivo T cells from subjects with HIES failed to produce IL-17, but not IL-2, tumour-necrosis factor or interferon-gamma, on mitogenic stimulation with staphylococcal enterotoxin B or on antigenic stimulation with Candida albicans or streptokinase.
Westra et al. (2000)interferon-gammaT cellsCulture supernatants of PB T cells stimulated with recombinant gH(t(His)):gL contained high levels of interferon-gamma and no detectable interleukin-4, indicating their Th1 phenotype.
Aranami et al. (2002)interferon-gammaT cellsIn spite of the vigorous proliferation and expression of several activation markers, these SMLR-activated CD8(+) T cells hardly killed syngeneic targets and most of the CD8(+) T cells produced no interferon-gamma upon restimulation with DC.
Barnaba et al. (1994)IFN-gammaT cellHowever, all of the CD4+CD56+ T cell clones produced IFN-gamma but not IL-4 and IL-5 (Th1-like cell clones).
Brown et al. (1993)gamma interferonT cellsBiological assays and Northern (RNA) blot analyses for cytokines revealed that following activation with concanavalin A, T-cell clones reactive against the two Bb-1A epitopes produced interleukin-2, gamma interferon, and tumor necrosis factors beta and alpha, but not interleukin-4, suggesting that the Bb-1 antigen preferentially stimulates the Th1 subset of CD4+ T cells in cattle.
Amano et al. (2005)IFN-gammaT cellsThese decreases were accompanied by significantly lower numbers of alloreactive T cells developing to IFN-gamma-, but not IL-4-producing cells in the CCR5(-/-) recipients, suggesting suboptimal priming of T cells in the knockout recipients.
Spinozzi et al. (1997)IFN-gammaT lymphocytesHowever, despite high expression of this molecule, cloned allergen-specific cord CD4+ T lymphocytes were unable to produce IFN-gamma and/or IL-4.
Zloza et al. (2003)interferon gammaT cellsWe demonstrate here that the activated CD4dimCD8bright T cells are not undergoing apoptosis and do not produce significant intracellular levels of interferon gamma (IFNgamma), interleukin 2 (IL-2), or IL-10 but express elevated levels of intracellular IL-4 in comparison to CD8+CD4- and CD4+ T cells.
Feske et al. (1996)interferon-gammaT cellsFurthermore both childrens' T cells were unable to produce the cytokines IL-2, interferon-gamma (IFN-gamma), IL-4 and tumor necrosis factor-alpha (TNF-alpha).
Corrigan et al. (1995)IFN-gammaT-LCComparing the asthmatics with the controls, elevated percentages of CD4 T-LC expressed mRNA encoding IL-5, IL-4, and GM-CSF (P < 0.02) but not IL-3, IL-2, or IFN-gamma.
Kondo et al. (2004)interferon-gammaT cellT cell lines from patients with chronic hepatitis B had a lower level of proliferation (0- to 24.9-fold expansion by in vitro stimulation) and a higher ability to produce interferon-gamma (0-84% except for S89), while perforin-positive cells showed low frequencies (0-50% except for S89).
Aklilu et al. (2004)interferon-gammaT cellsExpanded T cells produced interferon-gamma, but not IL-4.
Komata et al. (2003)interferon-gammaT cellsWhen CD44low CD4+ T cells were activated with immobilized anti-CD3 antibody and interleukin-2, they proliferated and produced interferon-gamma but not interleukin-4.
Karenko et al. (2001)interferon-gammaT cellsChromosomally clonal T cells in the skin, blood, or lymph nodes of two Sezary syndrome patients express CD45RA, CD45RO, CDw150, and interleukin-4, but no interleukin-2 or interferon-gamma.
Murata et al. (2002)interferon-gamma mRNAT cellsInterleukin-4 (IL-4) and IL-10 mRNA were highly expressed on intrarenal T cells, while interferon-gamma mRNA was not detected.
Jenkins et al. (1987)interferon-gammaT-cellT cells stimulated in this manner failed to produce IL-2, but interleukin 3, interferon-gamma, and IL-2 receptors were partially induced and T-cell receptor beta mRNA was fully induced.