Viewing negative mentions of gene expression of cd3g (X. laevis) in T cells

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Pouderoux et al. (1997)CD3T-cellImmunohistochemistry in paraffin-embedded tissue sections was positive for the pan T-cell marker MTI, weakly positive for UCHLI, and negative for CD3, and B-cell markers were negative; these findings were consistent with the diagnosis of T-cell lymphoma.
Xu et al. (2002)CD3T-cellThe recent biopsy of the cervical node showed typical features of AILT Flow cytometric immunophenotyping identified an aberrant CD4+ T-cell population that lacked surface CD3.
van de Griend et al. (1987)CD3T cellThis is suggested by the finding that the CD3+4-8- clones do virtually not express the common epitope of the T cell receptor alpha, beta-chains as identified by the WT31 MAb.
Watabe et al. (2002)CD3T cellImmunohistochemical staining revealed that the large cells were positive for the B cell marker, CD20, but negative for the T cell marker, CD3.
Kitzmann et al. (2008)CD3T-cellThe histopathology showed diffuse infiltration with a clonal population of lymphocytic cells that were CD3 positive and CD20 negative, consistent with T-cell NHL.
Patey-Mariaud De Serre et al. (2000)CD3T-lymphocytesAIMS: We recently showed that refractory sprue is distinct from coeliac disease, the former being characterized by abnormal intraepithelial T-lymphocytes expressing a cytoplasmic CD3 chain (CD3c), lacking CD3 and CD8 surface expression, and showing TCRgamma gene rearrangements.
Mingari et al. (1987)CD3T-cellSince previous studies indicated that PHA may be inefficient in inducing lymphokine production by T-cell variants lacking the CD3/T cell receptor complex (TCR), CD3- clones were further stimulated with the calcium ionophore A23187 plus phorbol 12-myristate 13-acetate (PMA).
Bank et al. (1998)CD3T cellsRESULTS: Flow cytometric and immunohistochemical analysis of the PBMCs revealed a major population (consisting of approximately 85% of the CD4+ T cells) that lacked expression of CD3 and T-cell receptors on the cell surface (CD4+CD3- T cells), but did express CD3 peptides in the cytoplasm.
La Perle et al. (1998)CD3T-cellImmunohistochemically, the neoplastic lymphocytes expressed CD3 but not CD20 or kappa and lambda light chains, supporting a diagnosis of T-cell lymphosarcoma.
Spencer et al. (1989)CD3T cellsApproximately 20% of CD7+ cells (T cells and null cells) do not express CD4 or CD8 and 14% of the CD7+ cells do not express CD3 and are therefore not T cells.
Rajwanshi et al. (1997)CD3T cellThe neoplastic cells were positive for CD20(L26-A Pan B marker) and negative for CD15(Leu M1), CD3(Ber H2) and pan T cell markers.
Bank et al. (2001)CD3T-cellsThree of four had elevated IgE, thrombotic manifestations and lung involvement (asthma and/or infiltrates), and one had deforming sero-negative arthritis of the hands. 66-95% of their peripheral T-cells expressed CD4 but not CD3 or TCR molecules on the cell surface membrane.
Bregman et al. (2005)CD3T-cellFlow cytometry performed on a subsequent subcutaneous nodule demonstrated an abnormal T-cell population with expression of CD3, CD8, CD56, and T-cell receptor alpha-beta, and no expression of CD4.
Tremblay et al. (2005)CD3T-cellMalignant fibrous histiocytoma was tentatively diagnosed histologically; however, the tumor cells subsequently were found to be negative for histiocytic (MAC 387, antitrypsin), T-cell (CD3), and B-lymphocyte (immunoglobulin light chains, Ly 5/CD45R) markers, and positive for glial fibrillary acidic protein, vimentin, and S-100.
Nomura et al. (2005)CD3T-cellImmunohistologically, the tumor cells from the skin lesion expressed CD2, cytoplasmic CD3, CD56, and T-cell intracellular antigen-1 (TIA-1), but not surface CD3, CD19, and TdT.
Lishner et al. (1994)CD3T-cellRESULTS: Immunophenotyping established that the cells were CD3 positive, CD4 negative, CD8 negative, T-cell receptor (TCR)-alpha/beta negative, and TCR-gamma/delta positive.
Ballas et al. (1990)CD3T cellThis patient has a dramatic persistent deficiency in her circulating "classic" NK cells (CD3-, CD16+, NKH1+) and a simultaneous persistent expansion of a normally minor lymphocyte cell subset (CD3+, CD4-, CD8-, NKH1+) that does not express the alpha beta heterodimer of the T cell receptor.
Mookerjee et al. (1989)CD3T-cellThe proliferating cells predominantly expressed the T-cell antigens (CD3, CD4 and CD8), but not antigens of natural killer (NK) cells, B cells or mononuclear phagocytic cells.
Poonawalla et al. (2005)CD3T-cellMarkers CD3, Epstein-Barr virus-encoded small RNA, and T-cell receptor g gene rearrangement were positive, with CD4, 8, 30, 56, and TIA-1 negative.
Aoyama et al. (2001)CD3T cellExamination for surface antigens on lymphoma cells revealed a unique phenotype, positive for CD3 and T cell receptor (TCR) gamma/delta, but negative for CD2.
Boy et al. (2010)CD3T cellsAll were negative for CD20 with reactive T cells detected with CD3.
Plevyak et al. (2002)CD3T cellsThe majority of alphabeta and gammadelta positive T cells in both groups coexpressed the natural killer (NK) cell marker CD56, but lacked cell surface expression of CD3.
Tam et al. (1999)CD3T-cellFACS analysis showed that CD8+ cells did not express T-cell markers (CD3, TCRalphabeta or TCRgammadelta) or the NK-cell marker (NKR-P1).
Tsang et al. (1994)CD3T-cellTen cases showed EBER positivity, and all but one of them were negative for CD3 and other T-cell markers, except CD2.
Srour et al. (1988)CD3T cellsFour colour flow cytometric analysis revealed a small subset of T cells positive for CD3 and negative for CD5, CD4 and CD8.
Chia et al. (2009)CD3T-cellImmunohistochemical studies revealed that the tumour cells were positive for CD3, CD30, granzyme B and T-cell intracellular antigen-1, and negative for CD5 and CD56, with positive staining for Epstein-Barr virus (EBV) RNA on in situ hybridization.
Yachida et al. (2010)CD3T-cellOn immunohistochemistry, tumor cells were positive for CD3, CD4 and CD30, but negative for CD8, CD20 and CD56, implying a T-cell nature.
Okumura et al. (1992)CD3T cellDividing CD45RA-R0+ cells contain, therefore, many cells that have not yet expressed the CD3/T cell receptor complex and presumably have not yet undergone selective procedures.
Triebel et al. (1988)CD3T lymphocytesGiven the present results on the delta chain, it can be concluded that, in many individuals, a predominant fraction (V gamma 9+/V-AB12+) of circulating CD3+ TcR alpha/beta- T lymphocytes expresses a receptor with little, if any, combinatorial diversity.
Miller et al. (1992)CD3T-lymphocyteThe cultured cells did not express other antigens associated with T-lymphocyte (CD3, CD5, T-cell receptor [TCR] alpha/beta and TCR gamma/delta), B-lymphocyte (CD19), myeloid (MY8, CD33, and CD71), or monocytoid (CD14 and CD15) lineage and did not express the CD34 antigen associated with hematopoietic progenitors present on the starting population.
Sell et al. (1992)CD3T-cellThese suppressor cells expressed CD3, CD8, CD45RO, and the alpha, beta T-cell receptor, but not CD4 or CD56.
Anderson et al. (1989)CD3T cellNK cells do not express cell-surface CD3, or any known target recognition structure analogous to the T cell antigen receptor (TCR) heterodimers (alpha beta or gamma delta).
Sakata et al. (1990)CD3T cellThis disease include T-GL having CD3 antigen which forms a complex with T cell Ag receptor (TCR-alpha beta), and NK-GL which is CD3-negative but CD16- or NKH-1-positive, having non-MHC-restricted cytotoxicity.
Kurtin and Roche (1993)CD3T-cellThe phenotype of L-26 negative and CD3 or UCHL-1 positive accurately predicted T-cell phenotype in 95% (60 of 63) of the T-cell lymphomas and was not seen in any of the cases of B-cell lymphoma.
Thakar et al. (2006)CD3T cellsThe gating on CD3+ T cells removes the monocytes (expressing CD4 but not CD3 on their cell surface) from the gate.
Lee et al. (1995)CD3T-cellThe large cell immunoblastic lymphoma was of T-cell lineage, positive for the CD45RB, CD3, CD45RO, and CD43 antigens, and negative for the CD20 and CDw75 antigens.
Miyanishi and Ohno (1992)CD3T-cellThe SMZ-1 cells, as well as the parental lymphoma cells, were of helper/inducer T-cell immunophenotype; they were positive for CD2, CD3, and CD4 antigens, and negative for CD8.
Nichols et al. (1994)CD3T-cellIn contrast, the other case of lymphoproliferative disorder of granular lymphocytes rapidly evolved into an aggressive NK-cell lymphoma which did not express CD3, did express CD56, had germline T-cell receptor gene configurations, and had multiple clonal chromosomal abnormalities.
Tsutsumi et al. (1997)CD3T cellT cell receptors (TCR), surface CD3, CD4, CD5 and CD8 were not expressed.
Schroers et al. (2005)CD3T-cellComprehensive immunophenotypic analysis showed that the MCL cells expressed the typical B-lymphocytic markers, were CD5 and CD8 positive, but did not express other T-cell proteins, such as CD2, CD3, CD4, CD7, TCRalphabeta and TCRgammadelta.
Laveaux et al. (2010)CD3T cellBut the following data support the diagnosis of an HTLV1-related T cell lymphoma: i) All tumor cells were CD3+; ii) All were CD79a negative; iii) Our patient’s cutaneous mite infestation is specific to HTLV1 lymphomas; vi) There was a high level of circulating CD25+ cells in the remission blood specimen.
Chuang et al. (2001)CD3T-cellThese neoplastic cells expressed CD30, CD43, granzyme B and T-cell intracellular antigen-1, but not ALK1, CD3, CD20, CD45, CD79a, cytokeratin, and EMA.
Liu et al. (2003)CD3T-cellHistologic and immunohistochemical studies of a stereotactic biopsy of the mass showed a T-cell lymphoproliferative lesion positive for CD3, CD8, CD57, and T-cell intracellular antigen 1 and negative for CD4, CD56, CD30, anaplastic lymphoma kinase, and CD20.
Hassan et al. (2006)CD3T-cellNeoplastic cells had a CD45RO+, CD2+, CD7+, CD3+, CD5-, CD8+, CD56+, perforin+, FasL-negative, T-cell receptor (TCR)alphabeta-negative, TCRgammadelta+ profile.
Mori et al. (1993)CD3T cellWe describe a case of T-chronic lymphocytic leukaemia (T-CLL) with monoclonal proliferation of CD3+4+8- T cell expressing the interleukin 2 receptor (IL-2R) beta chain without expressing the alpha chain.
Minegishi et al. (1988)CD3T-cellTHP-6 cells were positive for CD7 and CD5 antigens and terminal deoxynucleotidyl transferase, but negative for CD2, CD1, CD4, CD8, CD10, cytoplasmic and surface CD3 and HLA-DR antigens, suggesting a precursor T-cell line.
Chuang et al. (2002)CD3T-cellHis tumor showed extensive coagulative necrosis with angioinvasion by large lymphoma cells expressing CD2, CD8, CD16, CD43, CD45, CD45RO, CD56, T-cell intracellular antigen-1, and granzyme B, but not CD3, CD4, CD20, CD57, CD68, and betaF1.
Merup et al. (1994)CD3T-cellAnalysis of gene expression in cells with TcR beta gene rearrangement indicated production of truncated TcR beta transcripts but no expression of the T-cell markers CD3, CD4, CD8, TcR alpha beta or delta on the cell surface.
Tjon et al. (2010)CD3T cellRCD II, however, is associated with the presence of an aberrant IEL population that lacks surface T cell receptor (TCR)-CD3 expression, but contains intracellular CD3?
Osuji et al. (2005)CD3T cellsT-cell prolymphocytes did not express cytotoxic granule proteins or NK-cell markers, were CD5+, CD45RO+ like normal spleen T cells, were CD2+, CD3+, CD45+, CD43+, TCRbeta+, but CD25-, CD30-, ALK-1-, TRAP-, DBA44-, and TdT-.
Nitta et al. (1995)CD3T cellThe small cells stained faintly with anti-CD5 in two specimens, but were negative for the T cell specific markers, CD2, CD3, CD7, and CD8.
Schindler et al. (2008)CD3T cellsInterestingly, we found that efficient Nef-mediated downmodulation of TCR-CD3 was preserved only in those animals with high CD4+ T cells, but not in those with low CD4+ T cells [28].
Masuda et al. (1994)CD3T cellPMT-2Y cells are positive for CD2, CD3, CD4, CD25, T cell receptor alpha beta and HLA-DR, but negative for CD1, CD7, CD8, CD19 and CD20, indicating that the clone belongs to a helper/inducer subset of T cells.
Chang et al. (2010)CD3T-cellImmunostaining for CD3, CD6, CD8, CD20, CD30, T-cell intracellular antigen-1, myeloperoxidase, and terminal deoxynucleotidyl transferase was either weak or negative (Fig. 3).
Okada et al. (2003)CD3T-cellThe lymphoma cells were positive for CD2, CD3, CD5, CD8, and T-cell receptor (TCR) beta F1, but negative for CD4, CD19, CD20, CD103, and CD56.
Bobryshev et al. (1998)CD3T-cellsT-cells (CD3+) and endothelial cells (von Willebrand factor+) were also negative for E-cadherin.
Kita et al. (1993)CD3T-cellLeukemic cells from all the patients were negative for T-cell-specific antigens, surface CD3, and T-cell-receptor molecules.
Robertson et al. (1996)CD3T cellNKL cells express CD2, CD6, CD11a, CD26, CD27, CD29, CD38, CD43, CD58, CD81, CD94, CD95, class II MHC, and the C1.7.1 antigen, but do not express detectable levels of CD3, CD4, CD5, CD8, CD14, CD19, CD20, CD28, alpha/beta or gamma/delta T cell receptors on the cell surface.
Scott et al. (1994)CD3T-cellAlthough two cases expressed CD4, no case expressed CD2, CD3, or CD8 and no case showed clonal rearrangement of genes encoding the T-cell receptor (TCR beta, gamma, delta).
Campo et al. (2006)CD3T-cellNinety-six percent of ALK+ and 40% of ALK– anaplastic large-cell lymphomas also lacked expression of the CD3 antigen, compared with 29% of peripheral T-cell lymphomas NOS and 20% of AILT.
Stranneheim et al. (2009)CD3T cellsB lymphocytes were isolated from tonsils by negative AutoMACS selection and elimination of T cells expressing the CD3+ surface receptor.
Varga et al. (1990)CD3T-cellIn all cases of LyP most larger cells expressed the activation antigen Ki-1 (CD30) and lacked expression of the T-cell antigen CD7 and at least one other T-cell antigen (CD2, CD3, CD5).
Palacios and Samaridis (1992)CD3-gammaT lymphocytesIndeed, both pro-B and pre-B clones can generate in vitro and in vivo B lymphocytes but not T lymphocytes; moreover, these clones do not express the CD3-gamma T-cell-specific gene, nor do they have rearranged gamma, delta, or beta T-cell antigen receptor genes.