| Document |
Target |
Regulator |
Anatomy |
Sentence |
| Gallis et al. (1986) | IL-2 | | | The IL-2 receptor expressed by these cells was not phosphorylated in the presence of PMA. |
| Decker et al. (2010) | IL-2 | | | Addition of IL-2 to activated B-CLL cells caused rapid phosphorylation of Akt, while IL-2 failed to significantly phosphorylate Akt in resting B-CLL cells. |
| Musso et al. (1995) | IL-2 | | granulocyte | Moreover, JAK3 became tyrosine phosphorylated in response to IL-2, IL-4, and IL-7 but not response to IFN-gamma or granulocyte/macrophage colony-stimulating factor. |
| Zu et al. (1990) | IL 2 | | | After cell conversion from an IL 2 dependent state to an IL 2 independent state, one of the five phosphoproteins, the 65-kDa protein, became constitutively phosphorylated even without addition of IL 2. |
| Candotti et al. (1997) | IL-2-mediated | | | Accordingly, IL-2-mediated phosphorylation of STAT5 was also absent or barely detectable. |
| Mills et al. (1992) | IL-2 | | | Despite the absence of LCK or FYN, IL-2 induced similar patterns of rapid tyrosine phosphorylation. |
| Musso et al. (1995) | IL-2 | | macrophage | Moreover, JAK3 became tyrosine phosphorylated in response to IL-2, IL-4, and IL-7 but not response to IFN-gamma or granulocyte/macrophage colony-stimulating factor. |
| Wadhwa et al. (2000) | IL-2 | | stem cell | We found no neutralization of granulocyte colony-stimulating factor, macrophage colony-stimulating factor, stem cell factor, IL-1beta, IL-2, IL-3, IL-4, IL-6, IL-9, IL-10, IL-12, tumour necrosis factor-alpha, oncostatin M (OSM) and IFN-gamma. |
| Gilmour et al. (2001) | IL-2-induced | | | Nine had no detectable gammac, four had abnormal gammac expression and no IL-2-induced JAK3 tyrosine phosphorylation, and one had normal gammac expression but no IL-2-induced JAK3 tyrosine phosphorylation, although JAK3 was present. |
| Jensen et al. (2001) | IL-2 | | | APC pulsed with S(72)(Tn), which differs from T(72)(Tn) by not having a methyl group in the serine amino acid side chain to which the glycan is attached, induced partial tyrosine phosphorylation of the TCR-zeta 21-kDa protein, no tyrosine phosphorylation of the MAP kinases and no IL-2 production. |
| Bright et al. (1997) | IL-2 | | T cells | We show here that treatment of activated T cells with TGF-beta inhibited IL-2-induced tyrosine phosphorylation and activation of Jak-1 and Stat 5 but not Jak-3 and Stat 3. |
after the article reference.